Edgar T. Walters, Ph.D.

Affiliations: 
1982- Integrative Biology and Pharmacology University of Texas Health Science Center at Houston, Houston, TX, United States 
Area:
Nociceptors, pain, memory, evolution, spinal cord injury, cell signaling
Website:
https://med.uth.edu/ibp/faculty/edgar-t-walters/
Google:
"Edgar Walters"
Mean distance: 12.42 (cluster 6)
 
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Publications

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Odem MA, Bavencoffe AG, Cassidy RM, et al. (2018) Isolated Nociceptors Reveal Multiple Specializations for Generating Irregular Ongoing Activity Associated with Ongoing Pain. Pain
Luo J, Bavencoffe A, Yang P, et al. (2017) Zinc inhibits TRPV1 to alleviate chemotherapy-induced neuropathic pain. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience
Wu Z, Li L, Xie F, et al. (2017) Activation of KCNQ channels suppresses spontaneous activity in DRG neurons and reduces chronic pain after spinal cord injury. Journal of Neurotrauma
Hu X, Adebiyi MG, Luo J, et al. (2016) Sustained Elevated Adenosine via ADORA2B Promotes Chronic Pain through Neuro-immune Interaction. Cell Reports
Oshima M, von Treuheim TD, Carroll J, et al. (2016) Peripheral injury alters schooling behavior in squid, Doryteuthis pealeii. Behavioural Processes
Bavencoffe A, Li Y, Wu Z, et al. (2016) Persistent Electrical Activity in Primary Nociceptors after Spinal Cord Injury Is Maintained by Scaffolded Adenylyl Cyclase and Protein Kinase A and Is Associated with Altered Adenylyl Cyclase Regulation. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 36: 1660-8
Luo J, Feng J, Liu S, et al. (2015) Erratum to: Molecular and cellular mechanisms that initiate pain and itch. Cellular and Molecular Life Sciences : Cmls
Boyette-Davis JA, Walters ET, Dougherty PM. (2015) Mechanisms involved in the development of chemotherapy-induced neuropathy. Pain Management. 5: 285-96
Luo J, Feng J, Liu S, et al. (2015) Molecular and cellular mechanisms that initiate pain and itch. Cellular and Molecular Life Sciences : Cmls
Yang Q, Wu Z, Hadden JK, et al. (2014) Persistent pain after spinal cord injury is maintained by primary afferent activity. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 34: 10765-9
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