Anni Seungeun Lee, B.S.

Affiliations: 
Weill Cornell Medical College, New York, NY, United States 
Area:
learning and memory
Google:
"Anni Lee"
Mean distance: 17.97 (cluster 17)
 
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Publications

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Baker MR, Lee AS, Rajadhyaksha AM. (2023) L-type calcium channels and neuropsychiatric diseases: Insights into genetic risk variant-associated genomic regulation and impact on brain development. Channels (Austin, Tex.). 17: 2176984
Burgdorf CE, Schierberl KC, Lee AS, et al. (2017) Extinction of contextual cocaine memories requires Cav1.2 within D1R-expressing cells and recruits hippocampal Cav1.2-dependent signaling mechanisms. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience
Kabir ZD, Lee AS, Burgdorf CE, et al. (2016) Cacna1c in the Prefrontal Cortex Regulates Depression-Related Behaviors via REDD1. Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology
Lee AS, De Jesús-Cortés H, Kabir ZD, et al. (2016) The Neuropsychiatric Disease-Associated Gene cacna1c Mediates Survival of Young Hippocampal Neurons. Eneuro. 3
Kabir ZD, Lee AS, Rajadhyaksha AM. (2016) L-type Ca(2+) channels in mood, cognition and addiction: Integrating human and rodent studies with a focus on behavioural endophenotypes. The Journal of Physiology
Lee AS, André JM, Pittenger C. (2014) Lesions of the dorsomedial striatum delay spatial learning and render cue-based navigation inflexible in a water maze task in mice. Frontiers in Behavioral Neuroscience. 8: 42
Quinn JJ, Pittenger C, Lee AS, et al. (2013) Striatum-dependent habits are insensitive to both increases and decreases in reinforcer value in mice. The European Journal of Neuroscience. 37: 1012-21
Lee AS, Gonzales KL, Lee A, et al. (2012) Selective genetic deletion of cacna1c in the mouse prefrontal cortex. Molecular Psychiatry. 17: 1051
Lee AS, Ra S, Rajadhyaksha AM, et al. (2012) Forebrain elimination of cacna1c mediates anxiety-like behavior in mice. Molecular Psychiatry. 17: 1054-5
Rajadhyaksha AM, Ra S, Kishinevsky S, et al. (2012) Behavioral characterization of cereblon forebrain-specific conditional null mice: a model for human non-syndromic intellectual disability. Behavioural Brain Research. 226: 428-34
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