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David Y. Huang, MD/PhD

Affiliations: 
2002- Neurology University of North Carolina, Chapel Hill, Chapel Hill, NC 
Area:
Stroke, Alzheimer's disease, neurodegeneration
Google:
"David Huang"
Mean distance: 14.8 (cluster 11)
 
SNBCP
Cross-listing: Alzheimer's Tree

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Publications

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Anderson LG, Meeker RB, Poulton WE, et al. (2010) Brain distribution of carboxy terminus of Hsc70-interacting protein (CHIP) and its nuclear translocation in cultured cortical neurons following heat stress or oxygen-glucose deprivation. Cell Stress & Chaperones. 15: 487-95
Strittmatter WJ, Burke JR, DeSerrano VS, et al. (1996) Protein: protein interactions in Alzheimer's disease and the CAG triplet repeat diseases. Cold Spring Harbor Symposia On Quantitative Biology. 61: 597-605
Montine TJ, Huang DY, Valentine WM, et al. (1996) Crosslinking of apolipoprotein E by products of lipid peroxidation. Journal of Neuropathology and Experimental Neurology. 55: 202-10
Roses AD, Einstein G, Gilbert J, et al. (1996) Morphological, biochemical, and genetic support for an apolipoprotein E effect on microtubular metabolism. Annals of the New York Academy of Sciences. 777: 146-57
Roses A, Saunders A, Pericak-Vance M, et al. (1996) Apolipoprotein is a susceptibility genetic locus that affects the expression of late-onset familial and sporadic Alzheimer's disease Biological Psychiatry. 39: 562
Roses AD, Saunders AM, Corder EH, et al. (1995) Influence of the susceptibility genes apolipoprotein E-epsilon 4 and apolipoprotein E-epsilon 2 on the rate of disease expressivity of late-onset Alzheimer's disease. Arzneimittel-Forschung. 45: 413-7
Huang DY, Weisgraber KH, Goedert M, et al. (1995) ApoE3 binding to tau tandem repeat I is abolished by tau serine262 phosphorylation. Neuroscience Letters. 192: 209-12
Huang DY, Weisgraber KH, Strittmatter WJ, et al. (1995) Interaction of apolipoprotein E with laminin increases neuronal adhesion and alters neurite morphology. Experimental Neurology. 136: 251-7
Strittmatter WJ, Weisgraber KH, Goedert M, et al. (1994) Hypothesis: microtubule instability and paired helical filament formation in the Alzheimer disease brain are related to apolipoprotein E genotype. Experimental Neurology. 125: 163-71; discussion 1
Sanan DA, Weisgraber KH, Russell SJ, et al. (1994) Apolipoprotein E associates with beta amyloid peptide of Alzheimer's disease to form novel monofibrils. Isoform apoE4 associates more efficiently than apoE3. The Journal of Clinical Investigation. 94: 860-9
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