Douglas A. Coulter
Affiliations: | University of Pennsylvania, Philadelphia, PA, United States |
Area:
Epilepsy ResearchWebsite:
http://stokes.chop.edu/web/coulter/staff.htmlGoogle:
"Douglas Coulter"Mean distance: 13.15 (cluster 6)
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Publications
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Adam CD, Schellinger ED, White A, et al. (2024) Chemogenetic Breakdown of the Dentate Gate Causes Seizures and Spatial Memory Deficits. Biorxiv : the Preprint Server For Biology |
Takano H, Hsu FC, Coulter DA. (2024) Prolonged Hyperactivity Elicits Massive and Persistent Chloride Ion Redistribution in Subsets of Cultured Hippocampal Dentate Granule Cells. Biorxiv : the Preprint Server For Biology |
Terzic B, Cui Y, Edmondson AC, et al. (2020) X-linked cellular mosaicism underlies age-dependent occurrence of seizure-like events in mouse models of CDKL5 deficiency disorder. Neurobiology of Disease. 105176 |
Kahn JB, Port RG, Anderson SA, et al. (2020) Modular, Circuit-Based Interventions Rescue Hippocampal-Dependent Social and Spatial Memory in a 22q11.2 Deletion Syndrome Mouse Model. Biological Psychiatry |
Mulcahey PJ, Tang S, Takano H, et al. (2020) Aged heterozygous Cdkl5 mutant mice exhibit spontaneous epileptic spasms. Experimental Neurology. 113388 |
Kahn JB, Port RG, Yue C, et al. (2019) Circuit-based interventions in the dentate gyrus rescue epilepsy-associated cognitive dysfunction. Brain : a Journal of Neurology |
Tang S, Terzic B, Wang IJ, et al. (2019) Altered NMDAR signaling underlies autistic-like features in mouse models of CDKL5 deficiency disorder. Nature Communications. 10: 2655 |
Wu Q, Takano H, Riddle DM, et al. (2019) Alpha-synuclein (αSyn) preformed fibrils induce endogenous αSyn aggregation, compromise synaptic activity and enhance synapse loss in cultured excitatory hippocampal neurons. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience |
Yun S, Reynolds RP, Petrof I, et al. (2018) Stimulation of entorhinal cortex-dentate gyrus circuitry is antidepressive. Nature Medicine |
He Z, Guo JL, McBride JD, et al. (2017) Amyloid-β plaques enhance Alzheimer's brain tau-seeded pathologies by facilitating neuritic plaque tau aggregation. Nature Medicine |