Joseph H. Neale
Affiliations: | Neuroscience | Georgetown University, Washington, DC |
Area:
Neuroscience Biology, Physiology Biology, Medical BiophysicsGoogle:
"Joseph Neale"Mean distance: 53433
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Publications
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Neale JH, Yamamoto T. (2019) N-acetylaspartylglutamate (NAAG) and Glutamate Carboxypeptidase II: An abundant peptide neurotransmitter-enzyme system with multiple clinical applications. Progress in Neurobiology. 101722 |
Neale JH, Olszewski R. (2019) A role for N-acetylaspartylglutamate (NAAG) and mGluR3 in cognition. Neurobiology of Learning and Memory. 158: 9-13 |
Nonaka T, Yamada T, Ishimura T, et al. (2017) A role for the locus coeruleus in the analgesic efficacy of N-acetylaspartylglutamate peptidase (GCPII) inhibitors ZJ43 and 2-PMPA. Molecular Pain. 13: 1744806917697008 |
Olszewski RT, Janczura KJ, Bzdega T, et al. (2017) NAAG Peptidase Inhibitors Act via mGluR3: Animal Models of Memory, Alzheimer's, and Ethanol Intoxication. Neurochemical Research |
Cao Y, Gao Y, Xu S, et al. (2016) Glutamate carboxypeptidase II gene knockout attenuates oxidative stress and cortical apoptosis after traumatic brain injury. Bmc Neuroscience. 17: 15 |
Gao Y, Xu S, Cui Z, et al. (2015) Mice lacking glutamate carboxypeptidase II develop normally, but are less susceptible to traumatic brain injury. Journal of Neurochemistry. 134: 340-53 |
Wenqiang C, Lonskaya I, Hebron ML, et al. (2014) Parkin-mediated reduction of nuclear and soluble TDP-43 reverses behavioral decline in symptomatic mice. Human Molecular Genetics. 23: 4960-9 |
Janczura KJ, Olszewski RT, Bzdega T, et al. (2013) NAAG peptidase inhibitors and deletion of NAAG peptidase gene enhance memory in novel object recognition test. European Journal of Pharmacology. 701: 27-32 |
Walder KK, Ryan SB, Bzdega T, et al. (2013) Immunohistological and electrophysiological evidence that N-acetylaspartylglutamate is a co-transmitter at the vertebrate neuromuscular junction. The European Journal of Neuroscience. 37: 118-29 |
Yamada T, Zuo D, Yamamoto T, et al. (2012) NAAG peptidase inhibition in the periaqueductal gray and rostral ventromedial medulla reduces flinching in the formalin model of inflammation. Molecular Pain. 8: 67 |