Rolf W. Stottmann, Ph.D.

Affiliations: 
2004-2011 Brigham and Women's Hospital, Harvard Medical School, Boston, MA, United States 
 2011- Pediatrics Cincinnati Children's Hospital Medical Center, Cincinnati, OH, United States 
Area:
cortical development, genetics
Website:
http://www.cincinnatichildrens.org/research/divisions/h/genetics/labs/stottmann/default/
Google:
"Rolf Stottmann"
Mean distance: 18.54 (cluster 32)
 
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Publications

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Bittermann E, Abdelhamed Z, Liegel RP, et al. (2019) Differential requirements of tubulin genes in mammalian forebrain development. Plos Genetics. 15: e1008243
Driver AM, Shumrick C, Stottmann RW. (2017) Ttc21b Is Required in Bergmann Glia for Proper Granule Cell Radial Migration. Journal of Developmental Biology. 5
DiStasio A, Driver A, Sund K, et al. (2017) Copb2 is essential for embryogenesis and hypomorphic mutations cause human microcephaly. Human Molecular Genetics
Li Z, Peng Y, Hufnagel RB, et al. (2017) Loss of SLC25A46 causes neurodegeneration by affecting mitochondrial dynamics and energy production in mice. Human Molecular Genetics. 26: 3776-3791
Snedeker J, Schock EN, Struve JN, et al. (2017) Unique spatiotemporal requirements for intraflagellar transport genes during forebrain development. Plos One. 12: e0173258
Driver AM, Kratz LE, Kelley RI, et al. (2016) Altered cholesterol biosynthesis causes precocious neurogenesis in the developing mouse forebrain. Neurobiology of Disease
Cionni M, Menke C, Stottmann RW. (2015) Novel genetic tools facilitate the study of cortical neuron migration. Mammalian Genome : Official Journal of the International Mammalian Genome Society
Jeruschke S, Jeruschke K, DiStasio A, et al. (2015) Everolimus Stabilizes Podocyte Microtubules via Enhancing TUBB2B and DCDC2 Expression. Plos One. 10: e0137043
Menke C, Cionni M, Siggers T, et al. (2015) Grhl2 is required in nonneural tissues for neural progenitor survival and forebrain development. Genesis (New York, N.Y. : 2000)
Saal HM, Prows CA, Guerreiro I, et al. (2015) A mutation in FRIZZLED2 impairs Wnt signaling and causes autosomal dominant omodysplasia. Human Molecular Genetics. 24: 3399-409
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