Severn B. Churn
Affiliations: | Neuroscience | Virginia Commonwealth University, Richmond, VA, United States |
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"Severn Churn"Mean distance: 106866
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| Deshpande LS, DeLorenzo RJ, Churn SB, et al. (2020) Neuronal-Specific Inhibition of Endoplasmic Reticulum Mg/Ca ATPase Ca Uptake in a Mixed Primary Hippocampal Culture Model of Status Epilepticus. Brain Sciences. 10 |
| Campbell JN, Gandhi A, Singh B, et al. (2014) Traumatic Brain Injury Causes a Tacrolimus-Sensitive Increase in Non-Convulsive Seizures in a Rat Model of Post-Traumatic Epilepsy. International Journal of Neurology & Brain Disorders. 1: 1-11 |
| Campbell JN, Low B, Kurz JE, et al. (2012) Mechanisms of dendritic spine remodeling in a rat model of traumatic brain injury. Journal of Neurotrauma. 29: 218-34 |
| Campbell JN, Register D, Churn SB. (2012) Traumatic brain injury causes an FK506-sensitive loss and an overgrowth of dendritic spines in rat forebrain. Journal of Neurotrauma. 29: 201-17 |
| Campbell JN, Gandhi A, Singh B, et al. (2011) Acute administration of tacrolimus after traumatic brain injury ameliorates post-traumatic epilepsy in a rat model F1000research. 2 |
| Reid WM, Rolfe A, Register D, et al. (2010) Strain-related differences after experimental traumatic brain injury in rats Journal of Neurotrauma. 27: 1243-1253 |
| Bracey JM, Kurz JE, Low B, et al. (2009) Prolonged seizure activity leads to increased Protein Kinase A activation in the rat pilocarpine model of status epilepticus. Brain Research. 1283: 167-76 |
| Blair RE, Sombati S, Churn SB, et al. (2008) Epileptogenesis causes an N-methyl-d-aspartate receptor/Ca2+-dependent decrease in Ca2+/calmodulin-dependent protein kinase II activity in a hippocampal neuronal culture model of spontaneous recurrent epileptiform discharges. European Journal of Pharmacology. 588: 64-71 |
| Kurz JE, Moore BJ, Henderson SC, et al. (2008) A cellular mechanism for dendritic spine loss in the pilocarpine model of status epilepticus. Epilepsia. 49: 1696-710 |
| Singleton MW, Holbert WH, Lee AT, et al. (2005) Modulation of CaM kinase II activity is coincident with induction of status epilepticus in the rat pilocarpine model Epilepsia. 46: 1389-1400 |