Martha C. Bohn

Affiliations: 
Pediatrics: Neurobiology Northwestern University, Evanston, IL 
Area:
Parkinson's Disease
Google:
"Martha Bohn"
Mean distance: 17.85 (cluster 40)
 
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Publications

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Jara JH, Stanford MJ, Zhu Y, et al. (2015) Healthy and diseased corticospinal motor neurons are selectively transduced upon direct AAV2-2 injection into the motor cortex. Gene Therapy
Jara JH, Genç B, Cox GA, et al. (2015) Corticospinal Motor Neurons Are Susceptible to Increased ER Stress and Display Profound Degeneration in the Absence of UCHL1 Function. Cerebral Cortex (New York, N.Y. : 1991)
Khodr CE, Becerra A, Han Y, et al. (2014) Targeting alpha-synuclein with a microRNA-embedded silencing vector in the rat substantia nigra: positive and negative effects. Brain Research. 1550: 47-60
Malchenko S, Xie J, de Fatima Bonaldo M, et al. (2014) Onset of rosette formation during spontaneous neural differentiation of hESC and hiPSC colonies. Gene. 534: 400-7
Rodríguez SS, Schwerdt JI, Barbeito CG, et al. (2013) Hypothalamic IGF-I gene therapy prolongs estrous cyclicity and protects ovarian structure in middle-aged female rats. Endocrinology. 154: 2166-73
Glavaski-Joksimovic A, Bohn MC. (2013) Mesenchymal stem cells and neuroregeneration in Parkinson's disease. Experimental Neurology. 247: 25-38
Jara JH, Villa SR, Khan NA, et al. (2012) AAV2 mediated retrograde transduction of corticospinal motor neurons reveals initial and selective apical dendrite degeneration in ALS. Neurobiology of Disease. 47: 174-83
Khodr CE, Pedapati J, Han Y, et al. (2012) Inclusion of a portion of the native SNCA 3'UTR reduces toxicity of human S129A SNCA on striatal-projecting dopamine neurons in rat substantia nigra. Developmental Neurobiology. 72: 906-17
Degeorge ML, Marlowe D, Werner E, et al. (2011) Combining glial cell line-derived neurotrophic factor gene delivery (AdGDNF) with L-arginine decreases contusion size but not behavioral deficits after traumatic brain injury. Brain Research. 1403: 45-56
Khodr CE, Sapru MK, Pedapati J, et al. (2011) An α-synuclein AAV gene silencing vector ameliorates a behavioral deficit in a rat model of Parkinson's disease, but displays toxicity in dopamine neurons. Brain Research. 1395: 94-107
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