Nikolaos K. Robakis
Affiliations: | Icahn School of Medicine at Mount Sinai, New York, NY, United States |
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"Nikolaos Robakis"Mean distance: 17.14 (cluster 3) | S | N | B | C | P |
Cross-listing: Alzheimer's Tree
Parents
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Sign in to add traineeMiguel A. Pappolla | grad student | ||
Geo Serban | grad student | 2005 | Mount Sinai School of Medicine |
Noel A. Warren | grad student | 2014 | Mount Sinai School of Medicine |
Zhao Xuan | grad student | 2014 | Mount Sinai School of Medicine |
Spiros Efthimiopoulos | post-doc | Mount Sinai School of Medicine |
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Publications
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Yoon Y, Voloudakis G, Doran N, et al. (2020) PS1 FAD mutants decrease ephrinB2-regulated angiogenic functions, ischemia-induced brain neovascularization and neuronal survival. Molecular Psychiatry |
Zhao J, Xiao Y, Liu X, et al. (2020) Substrate interaction inhibits γ-secretase production of amyloid-β peptides. Chemical Communications (Cambridge, England) |
Zhao J, Xiao Y, Liu X, et al. (2019) SUBSTRATE BINDING INHIBITS γ-SECRETASE CLEAVAGE OF AMYLOID PRECURSOR PROTEIN Alzheimers & Dementia. 15: 1595 |
Warren NA, Voloudakis G, Yoon Y, et al. (2018) The product of the γ-secretase processing of ephrinB2 regulates VE-cadherin complexes and angiogenesis. Cellular and Molecular Life Sciences : Cmls |
Huang Q, Voloudakis G, Ren Y, et al. (2017) Presenilin1/γ-secretase protects neurons from glucose deprivation-induced death by regulating miR-212 and PEA15. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology |
Fullard JF, Giambartolomei C, Hauberg ME, et al. (2017) Open chromatin profiling of human postmortem brain infers functional roles for non-coding schizophrenia loci. Human Molecular Genetics |
Nikolakopoulou AM, Georgakopoulos A, Robakis NK. (2016) Presenilin 1 promotes trypsin-induced neuroprotection via the PAR2/ERK signaling pathway. Effects of presenilin 1 FAD mutations. Neurobiology of Aging. 42: 41-9 |
Zhu L, Zhong M, Elder GA, et al. (2015) Phospholipid dysregulation contributes to ApoE4-associated cognitive deficits in Alzheimer's disease pathogenesis. Proceedings of the National Academy of Sciences of the United States of America. 112: 11965-70 |
Bruban J, Voloudakis G, Huang Q, et al. (2015) Presenilin 1 is necessary for neuronal, but not glial, EGFR expression and neuroprotection via γ-secretase-independent transcriptional mechanisms. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology |
Robakis NK, Georgakopoulos A. (2014) Allelic interference: a mechanism for trans-dominant transmission of loss of function in the neurodegeneration of familial Alzheimer's disease. Neuro-Degenerative Diseases. 13: 126-30 |