Giselle Cheung
Affiliations: | Physiology | University of Edinburgh, Edinburgh, Scotland, United Kingdom |
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Publications
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Cheung G, Cousin MA. (2019) Synaptic vesicle generation from activity-dependent bulk endosomes requires a dephosphorylation-dependent dynamin-syndapin interaction. Journal of Neurochemistry |
Ghézali G, Vasile F, Curry N, et al. (2019) Neuronal Activity Drives Astroglial Connexin 30 in Perisynaptic Processes and Shapes Its Functions. Cerebral Cortex (New York, N.Y. : 1991) |
Cheung G, Sibille J, Zapata J, et al. (2015) Activity-Dependent Plasticity of Astroglial Potassium and Glutamate Clearance. Neural Plasticity. 2015: 109106 |
Cheung G, Cousin MA. (2013) Synaptic vesicle generation from activity-dependent bulk endosomes requires calcium and calcineurin. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 33: 3370-9 |
Cheung G, Cousin MA. (2012) Adaptor protein complexes 1 and 3 are essential for generation of synaptic vesicles from activity-dependent bulk endosomes. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 32: 6014-23 |
Cheung G, Cousin MA. (2011) Quantitative analysis of synaptic vesicle pool replenishment in cultured cerebellar granule neurons using FM dyes. Journal of Visualized Experiments : Jove |
Kronenberg G, Gertz K, Cheung G, et al. (2010) Modulation of fate determinants Olig2 and Pax6 in resident glia evokes spiking neuroblasts in a model of mild brain ischemia. Stroke; a Journal of Cerebral Circulation. 41: 2944-9 |
Cheung G, Jupp OJ, Cousin MA. (2010) Activity-dependent bulk endocytosis and clathrin-dependent endocytosis replenish specific synaptic vesicle pools in central nerve terminals. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 30: 8151-61 |
Clayton EL, Sue N, Smillie KJ, et al. (2010) Dynamin I phosphorylation by GSK3 controls activity-dependent bulk endocytosis of synaptic vesicles. Nature Neuroscience. 13: 845-51 |
Färber K, Cheung G, Mitchell D, et al. (2009) C1q, the recognition subcomponent of the classical pathway of complement, drives microglial activation. Journal of Neuroscience Research. 87: 644-52 |