Eric G. Krause

Affiliations: 
University of Cincinnati, Cincinnati, OH 
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"Eric Krause"
Mean distance: 15.77 (cluster 19)
 
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Publications

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Mohammed M, Johnson DN, Wang LA, et al. (2021) Targeting angiotensin type 2 receptors located on pressor neurons in the nucleus of the solitary tract to relieve hypertension in mice. Cardiovascular Research
Brierley DI, Holt MK, Singh A, et al. (2021) Central and peripheral GLP-1 systems independently suppress eating. Nature Metabolism
Frazier CJ, Harden SW, Alleyne AR, et al. (2020) An Angiotensin-responsive Connection from the Lamina Terminalis to the Paraventricular Nucleus of the Hypothalamus Evokes Vasopressin Secretion to Increase Blood Pressure in Mice. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience
Elsaafien K, de Kloet AD, Krause EG, et al. (2020) Brain Angiotensin Type-1 and Type-2 Receptors in Physiological and Hypertensive Conditions: Focus on Neuroinflammation. Current Hypertension Reports. 22: 48
de Kloet AD, Cahill KM, Scott KA, et al. (2020) Overexpression of angiotensin converting enzyme 2 reduces anxiety-like behavior in female mice. Physiology & Behavior. 113002
Sharma RK, Oliveira AC, Yang T, et al. (2020) Gut Pathology and Its Rescue by ACE2 (Angiotensin-Converting Enzyme 2) in Hypoxia-Induced Pulmonary Hypertension. Hypertension (Dallas, Tex. : 1979). HYPERTENSIONAHA12014
Peris J, Steck M, Krause EG. (2020) Oxytocin treatment for alcoholism: Potential neurocircuitry targets. Neuropharmacology. 108091
Pati D, Harden SW, Sheng W, et al. (2020) Endogenous oxytocin inhibits hypothalamic corticotrophin-releasing hormone neurones following acute hypernatraemia. Journal of Neuroendocrinology. e12839
Sumners C, Alleyne A, Rodríguez V, et al. (2019) Brain angiotensin type-1 and type-2 receptors: cellular locations under normal and hypertensive conditions. Hypertension Research : Official Journal of the Japanese Society of Hypertension
Futch HS, McFarland KN, Moore BD, et al. (2019) An anti-CRF antibody suppresses the HPA axis and reverses stress-induced phenotypes. The Journal of Experimental Medicine
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