Elias Aizenman, Ph.D.

Affiliations: 
1989- Neurobiology University of Pittsburgh, Pittsburgh, PA, United States 
Area:
Zinc, Potassium Channels, Apoptosis, Redox, NMDA, TOPA
Website:
http://aizenmanlab.neurobio.pitt.edu/
Google:
"Elias Aizenman"
Mean distance: 13.23 (cluster 11)
 
SNBCP

Parents

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Chirstopher H. Price research assistant 1980-1981 Boston University
George  G. Bierkamper grad student 1981-1985 Johns Hopkins
Elise F. Stanley grad student 1981-1985 Johns Hopkins
Stuart A. Lipton post-doc 1985-1987 Harvard Medical School
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Publications

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XiangWei W, Kannan V, Xu Y, et al. (2019) Heterogeneous clinical and functional features of GRIN2D-related developmental and epileptic encephalopathy. Brain : a Journal of Neurology
Yeh CY, Ye Z, Moutal A, et al. (2019) Defining the Kv2.1-syntaxin molecular interaction identifies a first-in-class small molecule neuroprotectant. Proceedings of the National Academy of Sciences of the United States of America
Justice JA, Manjooran DT, Yeh CY, et al. (2018) Molecular neuroprotection induced by zinc-dependent expression of hepatitis C-derived protein NS5A targeting Kv2.1 potassium channels. The Journal of Pharmacology and Experimental Therapeutics
Yeh CY, Bulas AM, Moutal A, et al. (2017) Targeting a Potassium Channel/Syntaxin Interaction Ameliorates Cell Death in Ischemic Stroke. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience
Justice JA, Schulien AJ, He K, et al. (2017) Disruption of KV2.1 somato-dendritic clusters prevents the apoptogenic increase of potassium currents. Neuroscience
Ogden KK, Chen W, Swanger SA, et al. (2017) Molecular Mechanism of Disease-Associated Mutations in the Pre-M1 Helix of NMDA Receptors and Potential Rescue Pharmacology. Plos Genetics. 13: e1006536
Li D, Yuan H, Ortiz-Gonzalez XR, et al. (2016) GRIN2D Recurrent De Novo Dominant Mutation Causes a Severe Epileptic Encephalopathy Treatable with NMDA Receptor Channel Blockers. American Journal of Human Genetics
Kumar M, Reed N, Liu R, et al. (2016) Synthesis and Evaluation of Potent KCNQ2/3-specific Channel Activators. Molecular Pharmacology
Schulien AJ, Justice JA, Maio RD, et al. (2016) Zn(2+) -Induced Ca(2+) release via ryanodine receptors triggers calcineurin-dependent redistribution of cortical neuronal Kv2.1 K(+) channels. The Journal of Physiology
Aizenman E, Mastroberardino PG. (2015) Metals and neurodegeneration. Neurobiology of Disease. 81: 1-3
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