John P. Horn

Affiliations: 
University of Pittsburgh University of Pittsburgh, Pittsburgh, PA, United States 
Area:
Synaptic Integration
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"John Horn"
Mean distance: 11.83 (cluster 6)
 
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Publications

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Kullmann PH, Sikora KM, Clark KL, et al. (2016) HCN hyperpolarization-activated cation channels strengthen virtual nicotinic EPSPs and thereby elevate synaptic amplification in rat sympathetic neurons. Journal of Neurophysiology. jn.00223.2016
Springer MG, Kullmann PH, Horn JP. (2015) Virtual leak channels modulate firing dynamics and synaptic integration in rat sympathetic neurons: implications for ganglionic transmission in vivo. The Journal of Physiology. 593: 803-23
McCord MC, Kullmann PH, He K, et al. (2014) Syntaxin-binding domain of Kv2.1 is essential for the expression of apoptotic K+ currents. The Journal of Physiology. 592: 3511-21
Tucker KR, Huertas MA, Horn JP, et al. (2012) Pacemaker rate and depolarization block in nigral dopamine neurons: a somatic sodium channel balancing act. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 32: 14519-31
Ji H, Tucker KR, Putzier I, et al. (2012) Functional characterization of ether-à-go-go-related gene potassium channels in midbrain dopamine neurons - implications for a role in depolarization block. The European Journal of Neuroscience. 36: 2906-16
Norris CA, He K, Springer MG, et al. (2012) Regulation of neuronal proapoptotic potassium currents by the hepatitis C virus nonstructural protein 5A. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 32: 8865-70
Rimmer K, Horn JP. (2010) Weak and straddling secondary nicotinic synapses can drive firing in rat sympathetic neurons and thereby contribute to ganglionic amplification. Frontiers in Neurology. 1: 130
Kullmann PH, Horn JP. (2010) Vasomotor sympathetic neurons are more excitable than secretomotor sympathetic neurons in bullfrog paravertebral ganglia. Autonomic Neuroscience : Basic & Clinical. 155: 19-24
Kullmann PH, Horn JP. (2010) Homeostatic regulation of M-current modulates synaptic integration in secretomotor, but not vasomotor, sympathetic neurons in the bullfrog. The Journal of Physiology. 588: 923-38
Putzier I, Kullmann PH, Horn JP, et al. (2009) Cav1.3 channel voltage dependence, not Ca2+ selectivity, drives pacemaker activity and amplifies bursts in nigral dopamine neurons. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 29: 15414-9
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