David H. Gutmann

Affiliations: 
Biology and Biomedical Sciences (Neurosciences) Washington University, Saint Louis, St. Louis, MO 
Area:
Cell Biology, Oncology, Neuroscience Biology
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"David Gutmann"
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Publications

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Thumu SCR, Jain M, Soman S, et al. (2024) Correction: SRF-deficient astrocytes provide neuroprotection in mouse models of excitotoxicity and neurodegeneration. Elife. 13
Gutmann DH. (2024) Leveraging murine models of the neurofibromatosis type 1 cancer predisposition syndrome to elucidate the cellular circuits that drive pediatric low-grade glioma formation and progression. Neuro-Oncology Advances. 6: vdae054
Anastasaki C, Chatterjee J, Koleske JP, et al. (2024) NF1 mutation-driven neuronal hyperexcitability sets a threshold for tumorigenesis and therapeutic targeting of murine optic glioma. Neuro-Oncology
Thumu SCR, Jain M, Soman S, et al. (2024) SRF-deficient astrocytes provide neuroprotection in mouse models of excitotoxicity and neurodegeneration. Elife. 13
Kuhrt LD, Motta E, Elmadany N, et al. (2023) Neurofibromatosis 1 (NF1) mutation results in impaired function of human induced pluripotent stem cell-derived microglia. Disease Models & Mechanisms
Tang Y, Gutmann DH. (2023) Neurofibromatosis Type 1-Associated Optic Pathway Gliomas: Current Challenges and Future Prospects. Cancer Management and Research. 15: 667-681
de Andrade Costa A, Chatterjee J, Cobb O, et al. (2021) Immune deconvolution and temporal mapping identifies stromal targets and developmental intervals for abrogating murine low-grade optic glioma formation. Neuro-Oncology Advances. 4: vdab194
Fisher MJ, Jones DTW, Li Y, et al. (2021) Integrated molecular and clinical analysis of low-grade gliomas in children with neurofibromatosis type 1 (NF1). Acta Neuropathologica
Jain M, Das S, Lu PPY, et al. (2021) SRF is required for maintenance of astrocytes in non-reactive state in the mammalian brain. Eneuro
Mo J, Anastasaki C, Chen Z, et al. (2020) Humanized neurofibroma model from induced pluripotent stem cells delineates tumor pathogenesis and developmental origins. The Journal of Clinical Investigation
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