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Giles E. Hardingham

Affiliations: 
University of Edinburgh, Edinburgh, Scotland, United Kingdom 
Area:
NMDA receptors
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"Giles Hardingham"
Mean distance: 15.85 (cluster 11)
 		SNBCP
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Parents

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Hilmar Bading post-doc 1994-2001 MRC-LMB

Children

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Kunal Gupta grad student 2008-2012

Collaborators

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Michelle M. Aarts collaborator University of Toronto
BETA: Related publications

Publications

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Booker SA, Simões de Oliveira L, Anstey NJ, et al. (2020) Input-Output Relationship of CA1 Pyramidal Neurons Reveals Intact Homeostatic Mechanisms in a Mouse Model of Fragile X Syndrome. Cell Reports. 32: 107988
Qiu J, Dando O, Febery JA, et al. (2020) Neuronal Activity and Its Role in Controlling Antioxidant Genes. International Journal of Molecular Sciences. 21
Hardingham GE. (2020) Targeting Synaptic NMDA Receptor Co-agonism as a Therapy for Alzheimer's Disease? Cell Metabolism. 31: 439-440
Zhao C, Devlin AC, Chouhan AK, et al. (2019) Mutant C9orf72 human iPSC-derived astrocytes cause non-cell autonomous motor neuron pathophysiology. Glia
Bas-Orth C, Schneider J, Lewen A, et al. (2019) The mitochondrial calcium uniporter is crucial for the generation of fast cortical network rhythms. Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism. 271678X19887777
Booker SA, Domanski APF, Dando OR, et al. (2019) Altered dendritic spine function and integration in a mouse model of fragile X syndrome. Nature Communications. 10: 4813
Hardingham G. (2019) NMDA receptor C-terminal signaling in development, plasticity, and disease. F1000research. 8
Marwick KFM, Skehel PA, Hardingham GE, et al. (2019) The human NMDA receptor GluN2A variant influences channel blocker potency. Pharmacology Research & Perspectives. 7: e00495
Marwick KFM, Hansen KB, Skehel PA, et al. (2019) Functional assessment of triheteromeric NMDA receptors containing a human variant associated with epilepsy. The Journal of Physiology
Johnstone M, Vasistha NA, Barbu MC, et al. (2018) Reversal of proliferation deficits caused by chromosome 16p13.11 microduplication through targeting NFκB signaling: an integrated study of patient-derived neuronal precursor cells, cerebral organoids and in vivo brain imaging. Molecular Psychiatry
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