David Cantu, Ph.D.
Affiliations: | 2010 | University of Colorado Anschutz Medical Campus, Denver, Aurora, CO |
Area:
Neuroscience Biology, Toxicology, Cell BiologyGoogle:
"David Cantu"Mean distance: 26716.5
Parents
Sign in to add mentor| Manisha Patel | grad student | 2010 | University of Colorado, Denver | |
| (Mitochondrial aconitase is a target and a source of reactive oxygen species: Implication for neurotoxicity.) | ||||
BETA: Related publications
See more...
Publications
|
You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect. |
| Zawada WM, Banninger GP, Thornton J, et al. (2011) Generation of reactive oxygen species in 1-methyl-4-phenylpyridinium (MPP+) treated dopaminergic neurons occurs as an NADPH oxidase-dependent two-wave cascade. Journal of Neuroinflammation. 8: 129 |
| Cantu D, Fulton RE, Drechsel DA, et al. (2011) Mitochondrial aconitase knockdown attenuates paraquat-induced dopaminergic cell death via decreased cellular metabolism and release of iron and H₂O₂. Journal of Neurochemistry. 118: 79-92 |
| Cantu D, Fulton R, Drechsel D, et al. (2010) Mitochondrial Aconitase Knockdown Decreases Cellular Metabolism and Prevents Paraquat-dependent Dopaminergic Neuronal Death Free Radical Biology and Medicine. 49: S154 |
| Cantu D, Schaack J, Patel M. (2009) Oxidative inactivation of mitochondrial aconitase results in iron and H2O2-mediated neurotoxicity in rat primary mesencephalic cultures. Plos One. 4: e7095 |