Christian Haass, PhD, University of Heidelberg 1989
Affiliations: | Biochemistry | Ludwig-Maximilians-Universität München, München, Bayern, Germany |
Area:
http://www.researchprofiles.collexis.com/jad/expert.asp?n=&u_id=25Website:
http://www.cipsm.de/en/aboutCIPSM/members/haassChristian/index.html?style=0Google:
"Christian Haass"Bio:
http://en.wikipedia.org/wiki/Christian_Haass
Mean distance: 17.66 (cluster 31) | S | N | B | C | P |
Cross-listing: Alzheimer's Tree - Chemistry Tree
Parents
Sign in to add mentor| Dennis J. Selkoe | post-doc | 1990-1992 | Brigham and Women's Hospital, Harvard Medical School |
Children
Sign in to add trainee| Laura T. Haas | research assistant | 2012-2012 | Universität München |
| Ulf Dettmer | grad student | (Chemistry Tree) | |
| Xianyuan Xiang | grad student | 2013- | Universität München |
| Stefan Prokop | grad student | 2002-2005 | |
| Dieter Edbauer | post-doc | LMU |
Collaborators
Sign in to add collaborator| Matthias Brendel | collaborator | ||
| Yuetiva Deming | collaborator | 2017- | Ludwig Maximilian University Munich |
BETA: Related publications
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Publications
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| Kolabas ZI, Kuemmerle LB, Perneczky R, et al. (2023) Distinct molecular profiles of skull bone marrow in health and neurological disorders. Cell |
| Johnson ECB, Bian S, Haque RU, et al. (2023) Cerebrospinal fluid proteomics define the natural history of autosomal dominant Alzheimer's disease. Nature Medicine |
| Finze A, Biechele G, Rauchmann BS, et al. (2023) Individual regional associations between Aβ-, tau- and neurodegeneration (ATN) with microglial activation in patients with primary and secondary tauopathies. Molecular Psychiatry |
| Hipke K, Pitter B, Hruscha A, et al. (2023) Loss of TDP-43 causes ectopic endothelial sprouting and migration defects through increased , and . Frontiers in Cell and Developmental Biology. 11: 1169962 |
| Gnörich J, Reifschneider A, Wind K, et al. (2023) Depletion and activation of microglia impact metabolic connectivity of the mouse brain. Journal of Neuroinflammation. 20: 47 |
| van Lengerich B, Zhan L, Xia D, et al. (2023) A TREM2-activating antibody with a blood-brain barrier transport vehicle enhances microglial metabolism in Alzheimer's disease models. Nature Neuroscience |
| Biel D, Suárez-Calvet M, Hager P, et al. (2023) sTREM2 is associated with amyloid-related p-tau increases and glucose hypermetabolism in Alzheimer's disease. Embo Molecular Medicine. e16987 |
| Paolicelli RC, Sierra A, Stevens B, et al. (2022) Microglia states and nomenclature: A field at its crossroads. Neuron. 110: 3458-3483 |
| Xiang X, Tahirovic S, Ziegler S, et al. (2022) Response to Comment on "Microglial activation states drive glucose uptake and FDG-PET alterations in neurodegenerative diseases". Science Translational Medicine. 14: eabn5104 |
| Xia D, Lianoglou S, Sandmann T, et al. (2022) Novel App knock-in mouse model shows key features of amyloid pathology and reveals profound metabolic dysregulation of microglia. Molecular Neurodegeneration. 17: 41 |