Marina V. Yasvoina, Ph.D.

Affiliations: 
2013 Neuroscience Northwestern University, Evanston, IL 
Area:
Nanoscience
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"Marina Yasvoina"
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Hande Ozdinler grad student 2013 Northwestern
 (eGFP expression under the Uchl1 promoter labels corticospinal motor neurons and a subpopulation of degeneration resistant spinal motor neurons in ALS mouse models.)
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Publications

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Guarascio R, Salih D, Yasvoina M, et al. (2019) Negative Regulator of Ubiquitin-Like Protein 1 modulates the autophagy-lysosomal pathway via p62 to facilitate the extracellular release of tau following proteasome impairment. Human Molecular Genetics
Gautam M, Jara JH, Sekerkova G, et al. (2016) Absence of alsin function leads to corticospinal motor neuron vulnerability via novel disease mechanisms. Human Molecular Genetics
Matarin M, Salih DA, Yasvoina M, et al. (2015) A genome-wide gene-expression analysis and database in transgenic mice during development of amyloid or tau pathology. Cell Reports. 10: 633-44
Yasvoina MV, Genç B, Jara JH, et al. (2013) eGFP expression under UCHL1 promoter genetically labels corticospinal motor neurons and a subpopulation of degeneration-resistant spinal motor neurons in an ALS mouse model. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 33: 7890-904
Srivastava DP, Woolfrey KM, Jones KA, et al. (2012) An autism-associated variant of Epac2 reveals a role for Ras/Epac2 signaling in controlling basal dendrite maintenance in mice. Plos Biology. 10: e1001350
Iordanescu G, Venkatasubramanian PN, Yasvoina MV, et al. (2008) IC-P3-185: A method to quantitate amyloid plaque load in a transgenic mouse model of Alzheimer's disease Alzheimer's & Dementia. 4: T81-T81
Zhao J, Fu Y, Yasvoina M, et al. (2007) Beta-site amyloid precursor protein cleaving enzyme 1 levels become elevated in neurons around amyloid plaques: implications for Alzheimer's disease pathogenesis. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 27: 3639-49
Ohno M, Cole SL, Yasvoina M, et al. (2007) BACE1 gene deletion prevents neuron loss and memory deficits in 5XFAD APP/PS1 transgenic mice. Neurobiology of Disease. 26: 134-45
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