Brie E. Paddock, Ph.D.
Affiliations: | Biology | Drexel University, Philadelphia, PA, United States |
Area:
Synaptic physiology, Alzheimer's DiseaseGoogle:
"Brie Paddock"Mean distance: 15.75 (cluster 11) | S | N | B | C | P |
Cross-listing: Alzheimer's Tree
Parents
Sign in to add mentorRavi Allada | research assistant | Northwestern | |
Sarah Smolik | research assistant | OHSU (FlyTree) | |
Noreen Reist | grad student | Colorado State | |
Daniel Marenda | post-doc | Drexel (FlyTree) |
BETA: Related publications
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Publications
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Mhatre SD, Satyasi V, Killen M, et al. (2014) Synaptic abnormalities in a Drosophila model of Alzheimer's disease. Disease Models & Mechanisms. 7: 373-85 |
Mhatre SD, Paddock BE, Saunders AJ, et al. (2013) Invertebrate models of Alzheimer's disease. Journal of Alzheimer's Disease : Jad. 33: 3-16 |
Saunders A, Utreja S, Paddock B, et al. (2012) P1-299: Calcineurin inhibition alters APP metabolism in vitro and in vivo Alzheimer's & Dementia. 8: P209-P209 |
Chakraborty R, Vepuri V, Mhatre SD, et al. (2011) Characterization of a Drosophila Alzheimer's disease model: pharmacological rescue of cognitive defects. Plos One. 6: e20799 |
Paddock BE, Wang Z, Biela LM, et al. (2011) Membrane penetration by synaptotagmin is required for coupling calcium binding to vesicle fusion in vivo. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 31: 2248-57 |
Paddock BE, Striegel AR, Hui E, et al. (2008) Ca2+-dependent, phospholipid-binding residues of synaptotagmin are critical for excitation-secretion coupling in vivo. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 28: 7458-66 |
Lin JM, Kilman VL, Keegan K, et al. (2002) A role for casein kinase 2alpha in the Drosophila circadian clock. Nature. 420: 816-20 |