Leslie Shinobu, MD PhD

Neurology Massachusetts General Hospital and Harvard University, Boston, MA, United States 
"Leslie Shinobu"
Mean distance: 17.48 (cluster 28)
BETA: Related publications


You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect.

Dai Y, Zheng K, Clark J, et al. (2014) Rapamycin drives selection against a pathogenic heteroplasmic mitochondrial DNA mutation. Human Molecular Genetics. 23: 637-47
Montine TJ, Shinobu L, Montine KS, et al. (2000) No difference in plasma or urinary F2-isoprostanes among patients with Huntington's disease or Alzheimer's disease and controls. Annals of Neurology. 48: 950
Andreassen OA, Ferrante RJ, Klivenyi P, et al. (2000) Partial deficiency of manganese superoxide dismutase exacerbates a transgenic mouse model of amyotrophic lateral sclerosis. Annals of Neurology. 47: 447-55
Mecocci P, Fanó G, Fulle S, et al. (1999) Age-dependent increases in oxidative damage to DNA, lipids, and proteins in human skeletal muscle. Free Radical Biology & Medicine. 26: 303-8
Ferrante RJ, Browne SE, Shinobu LA, et al. (1997) Evidence of increased oxidative damage in both sporadic and familial amyotrophic lateral sclerosis. Journal of Neurochemistry. 69: 2064-74
Ferrante RJ, Shinobu LA, Schulz JB, et al. (1997) Increased 3-nitrotyrosine and oxidative damage in mice with a human copper/zinc superoxide dismutase mutation. Annals of Neurology. 42: 326-34
Davis RE, Miller S, Herrnstadt C, et al. (1997) Mutations in mitochondrial cytochrome c oxidase genes segregate with late-onset Alzheimer disease. Proceedings of the National Academy of Sciences of the United States of America. 94: 4526-31
Neil WK, Shinobu LA, Beal MF, et al. (1997) OXIDATIVE INJURY IN THE SPINAL CORDS OF TRANSGENIC MICE EXPRESSING MUTANT HUMAN SUPEROXIDE DISMUTASE Journal of Neuropathology and Experimental Neurology. 56: 611
Brouillet EP, Shinobu L, McGarvey U, et al. (1993) Manganese injection into the rat striatum produces excitotoxic lesions by impairing energy metabolism. Experimental Neurology. 120: 89-94
See more...