Ayodeji A. Asuni

Affiliations: 
New York University, New York, NY, United States 
Area:
Neuroscience, Immunotherapy, Prion, Alzheimer's Disease
Google:
"Ayodeji Asuni"
Mean distance: 21.58 (cluster 51)
 
Cross-listing: Alzheimer's Tree

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Publications

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Pitera AP, Asuni AA, O'Connor V, et al. (2019) Pathogenic tau does not drive activation of the unfolded protein response. The Journal of Biological Chemistry
Asuni AA, Guridi M, Sanchez S, et al. (2015) Antioxidant peroxiredoxin 6 protein rescues toxicity due to oxidative stress and cellular hypoxia in vitro, and attenuates prion-related pathology in vivo. Neurochemistry International. 90: 152-65
Amin J, Paquet C, Baker A, et al. (2015) Effect of amyloid-β (Aβ) immunization on hyperphosphorylated tau: a potential role for glycogen synthase kinase (GSK)-3β. Neuropathology and Applied Neurobiology. 41: 445-57
Hallaq R, Volpicelli F, Cuchillo-Ibanez I, et al. (2015) The Notch intracellular domain represses CRE-dependent transcription. Cellular Signalling. 27: 621-9
West J, Dinh ND, Hardelauf H, et al. (2015) Bridging two cultures: Minimalistic networks prepared by microfl uidic arraying, and open access compartments for electrophysiology Neuromethods. 103: 39-56
Asuni AA, Pankiewicz JE, Sadowski MJ. (2014) Reply: To PMID 24687915. Annals of Neurology. 76: 630-1
Pankiewicz JE, Guridi M, Kim J, et al. (2014) Blocking the apoE/Aβ interaction ameliorates Aβ-related pathology in APOE ε2 and ε4 targeted replacement Alzheimer model mice. Acta Neuropathologica Communications. 2: 75
Asuni AA, Guridi M, Pankiewicz JE, et al. (2014) Modulation of amyloid precursor protein expression reduces β-amyloid deposition in a mouse model. Annals of Neurology. 75: 684-99
Asuni AA, Gray B, Bailey J, et al. (2014) Analysis of the hippocampal proteome in ME7 prion disease reveals a predominant astrocytic signature and highlights the brain-restricted production of clusterin in chronic neurodegeneration. The Journal of Biological Chemistry. 289: 4532-45
Kuszczyk MA, Sanchez S, Pankiewicz J, et al. (2013) Blocking the interaction between apolipoprotein E and Aβ reduces intraneuronal accumulation of Aβ and inhibits synaptic degeneration. The American Journal of Pathology. 182: 1750-68
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