Vahri Beaumont

Affiliations: 
University of California, Los Angeles, Los Angeles, CA 
Area:
synaptic transmission, signalling
Google:
"Vahri Beaumont"
Mean distance: 13.5 (cluster 32)
 
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Publications

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Aaronson J, Beaumont V, Blevins RA, et al. (2021) HDinHD: A Rich Data Portal for Huntington's Disease Research. Journal of Huntington's Disease
Stott AJ, Maillard MC, Beaumont V, et al. (2021) Evaluation of 5-(Trifluoromethyl)-1,2,4-oxadiazole-Based Class IIa HDAC Inhibitors for Huntington's Disease. Acs Medicinal Chemistry Letters. 12: 380-388
Chakroborty S, Manfredsson FP, Dec AM, et al. (2020) Phosphodiesterase 9A Inhibition Facilitates Corticostriatal Transmission in Wild-Type and Transgenic Rats That Model Huntington's Disease. Frontiers in Neuroscience. 14: 466
Perez-Rosello T, Gelman S, Tombaugh G, et al. (2019) Enhanced striatopallidal gamma-aminobutyric acid (GABA) receptor transmission in mouse models of huntington's disease. Movement Disorders : Official Journal of the Movement Disorder Society
Svedberg MM, Varnäs K, Varrone A, et al. (2019) In vitro phosphodiesterase 10A (PDE10A) binding in whole hemisphere human brain using the PET radioligand [F]MNI-659. Brain Research
Padovan-Neto FE, Jurkowski L, Murray C, et al. (2018) Age- and sex-related changes in cortical and striatal nitric oxide synthase in the Q175 mouse model of Huntington's disease. Nitric Oxide : Biology and Chemistry
Luckhurst CA, Aziz O, Beaumont V, et al. (2018) Development and characterization of a CNS-penetrant benzhydryl hydroxamic acid class IIa histone deacetylase inhibitor. Bioorganic & Medicinal Chemistry Letters
Khakh BS, Beaumont V, Cachope R, et al. (2017) Unravelling and Exploiting Astrocyte Dysfunction in Huntington's Disease. Trends in Neurosciences
Beaumont V, Zhong S, Lin H, et al. (2016) Phosphodiesterase 10A Inhibition Improves Cortico-Basal Ganglia Function in Huntington's Disease Models. Neuron
Beaumont V, Mrzljak L, Dijkman U, et al. (2016) The novel KMO inhibitor CHDI-340246 leads to a restoration of electrophysiological alterations in mouse models of Huntington's disease. Experimental Neurology
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