Tobias M. Boeckers, MD

Affiliations: 
University of Ulm, Ulm, Baden-Württemberg, Germany 
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"Tobias Boeckers"
Mean distance: 16.67 (cluster 32)
 
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Publications

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Urrutia-Ruiz C, Rombach D, Cursano S, et al. (2022) Deletion of the Autism-Associated Protein SHANK3 Abolishes Structural Synaptic Plasticity after Brain Trauma. International Journal of Molecular Sciences. 23
Mossa A, Pagano J, Ponzoni L, et al. (2021) Developmental impaired Akt signaling in the Shank1 and Shank3 double knock-out mice. Molecular Psychiatry
Culotta L, Scalmani P, Vinci E, et al. (2020) SULT4A1 Modulates Synaptic Development and Function by Promoting the Formation of PSD-95/NMDAR Complex. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 40: 7013-7026
Lutz AK, Pfaender S, Incearap B, et al. (2020) Autism-associated SHANK3 mutations impair maturation of neuromuscular junctions and striated muscles. Science Translational Medicine. 12
Ey E, Bourgeron T, Boeckers TM, et al. (2020) Editorial: Shankopathies: Shank Protein Deficiency-Induced Synaptic Diseases. Frontiers in Molecular Neuroscience. 13: 11
Cursano S, Battaglia CR, Urrutia-Ruiz C, et al. (2020) A CRHR1 antagonist prevents synaptic loss and memory deficits in a trauma-induced delirium-like syndrome. Molecular Psychiatry
Jesse S, Müller HP, Schoen M, et al. (2019) Severe white matter damage in SHANK3 deficiency: a human and translational study. Annals of Clinical and Translational Neurology
Picchiarelli G, Demestre M, Zuko A, et al. (2019) FUS-mediated regulation of acetylcholine receptor transcription at neuromuscular junctions is compromised in amyotrophic lateral sclerosis. Nature Neuroscience
Deshpande D, Higelin J, Schoen M, et al. (2019) Synaptic FUS Localization During Motoneuron Development and Its Accumulation in Human ALS Synapses. Frontiers in Cellular Neuroscience. 13: 256
Bursch F, Kalmbach N, Naujock M, et al. (2019) Altered calcium dynamics and glutamate receptor properties in iPSC derived motor neurons from ALS patients with C9orf72, FUS, SOD1 or TDP43 mutations. Human Molecular Genetics
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