Ryan E. Loy

Affiliations: 
Chromocell Corporation, North Brunswick Township, NJ, United States 
Area:
Therapeutics and Flavor Discovery
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"Ryan Loy"
Mean distance: 16.52 (cluster 46)
 
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Parents

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Robert T. Dirksen grad student 2005-2010 Rochester
 (Altered Type 1 Ryanodine Receptor Activity and Functional Rescue In a Mouse Model of Central Core Disease.)
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Publications

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Loy RE, Lueck JD, Mostajo-Radji MA, et al. (2012) Allele-specific gene silencing in two mouse models of autosomal dominant skeletal myopathy. Plos One. 7: e49757
Loy RE, Orynbayev M, Xu L, et al. (2011) Muscle weakness in Ryr1I4895T/WT knock-in mice as a result of reduced ryanodine receptor Ca2+ ion permeation and release from the sarcoplasmic reticulum. The Journal of General Physiology. 137: 43-57
Boncompagni S, Loy RE, Dirksen RT, et al. (2010) The I4895T mutation in the type 1 ryanodine receptor induces fiber-type specific alterations in skeletal muscle that mimic premature aging. Aging Cell. 9: 958-70
Zhou H, Lillis S, Loy RE, et al. (2010) Multi-minicore disease and atypical periodic paralysis associated with novel mutations in the skeletal muscle ryanodine receptor (RYR1) gene. Neuromuscular Disorders : Nmd. 20: 166-73
Loy RE, Mostajo-Radji MA, Lueck JD, et al. (2010) Allele Specific Gene Silencing in Autosomal-Dominant Skeletal Myopathies Biophysical Journal. 98: 712a-713a
Fallon JL, Baker MR, Xiong L, et al. (2009) Crystal structure of dimeric cardiac L-type calcium channel regulatory domains bridged by Ca2+* calmodulins. Proceedings of the National Academy of Sciences of the United States of America. 106: 5135-40
Loy RE, Apostol S, Orynbayev M, et al. (2009) Deficits in Ca2+ Release and in vivo Muscle Strength in Heterozygous I4895T RyR1 Knock-In Mice Biophysical Journal. 96: 236a
Zvaritch E, Depreux F, Kraeva N, et al. (2007) An Ryr1I4895T mutation abolishes Ca2+ release channel function and delays development in homozygous offspring of a mutant mouse line. Proceedings of the National Academy of Sciences of the United States of America. 104: 18537-42
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