Stefan Remy
Affiliations: | Neuronal Networks Group | German Center for Neurodegenerative Diseases (DZNE) |
Area:
Dendritic signal integration, Synaptic plasticity, Hippocampus, Theta Oscillations, LocomotionGoogle:
"Stefan Remy"Mean distance: 14.21 (cluster 6) | S | N | B | C | P |
Parents
Sign in to add mentorHeinz Beck | grad student | 2001-2005 | University of Bonn |
Nelson Spruston | post-doc |
Children
Sign in to add traineeCollaborators
Sign in to add collaboratorRoland Krueppel | collaborator | 2007-2010 | University of Bonn, Germany |
BETA: Related publications
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Publications
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Sosulina L, Mittag M, Geis HR, et al. (2021) Hippocampal hyperactivity in a rat model of Alzheimer's disease. Journal of Neurochemistry |
Bertan F, Wischhof L, Sosulina L, et al. (2020) Correction: Loss of Ryanodine Receptor 2 impairs neuronal activity-dependent remodeling of dendritic spines and triggers compensatory neuronal hyperexcitability. Cell Death and Differentiation |
Bertan F, Wischhof L, Sosulina L, et al. (2020) Loss of Ryanodine Receptor 2 impairs neuronal activity-dependent remodeling of dendritic spines and triggers compensatory neuronal hyperexcitability. Cell Death and Differentiation |
Dalügge D, Remy S. (2018) Human Cortical Dendrites: Stretched to Perform Better? Cell. 175: 635-637 |
Musial TF, Molina-Campos E, Bean LA, et al. (2018) Store depletion-induced h-channel plasticity rescues a channelopathy linked to Alzheimer's disease. Neurobiology of Learning and Memory |
Müller C, Remy S. (2017) Septo-hippocampal interaction. Cell and Tissue Research |
Justus D, Dalügge D, Bothe S, et al. (2016) Glutamatergic synaptic integration of locomotion speed via septoentorhinal projections. Nature Neuroscience |
Schmid LC, Mittag M, Poll S, et al. (2016) Dysfunction of Somatostatin-Positive Interneurons Associated with Memory Deficits in an Alzheimer's Disease Model. Neuron |
Müller C, Remy S. (2016) Slowly Building Excitement. Cell. 165: 1568-9 |
Wagner J, Krauss S, Shi S, et al. (2015) Reducing tau aggregates with anle138b delays disease progression in a mouse model of tauopathies. Acta Neuropathologica |