Maria Passafaro
Affiliations: | IN-CNR Milan |
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"Maria Passafaro"Mean distance: 17.07
Children
Sign in to add traineeLuca Murru | grad student | 2012-2018 | IN-CNR |
Lisa Mapelli | post-doc | 2010-2012 |
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Publications
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Miozzo F, Murru L, Maiellano G, et al. (2024) Disruption of the autism-associated gene leads to transcriptional alterations, synapse overgrowth, and defective network activity in the CA1. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience |
Alaverdian D, Corradi AM, Sterlini B, et al. (2023) Modelling PCDH19 clustering epilepsy by Neurogenin 2 induction of patient-derived induced pluripotent stem cells. Epileptic Disorders : International Epilepsy Journal With Videotape |
Giansante G, Mazzoleni S, Zippo AG, et al. (2023) Neuronal network activity and connectivity are impaired in a conditional knockout mouse model with PCDH19 mosaic expression. Molecular Psychiatry |
Moretto E, Miozzo F, Longatti A, et al. (2023) The tetraspanin TSPAN5 regulates AMPAR exocytosis by interacting with the AP4 complex. Elife. 12 |
Focchi E, Cambria C, Pizzamiglio L, et al. (2022) ATM rules neurodevelopment and glutamatergic transmission in the hippocampus but not in the cortex. Cell Death & Disease. 13: 616 |
Gerosa L, Mazzoleni S, Rusconi F, et al. (2022) The epilepsy-associated protein PCDH19 undergoes NMDA receptor-dependent proteolytic cleavage and regulates the expression of immediate-early genes. Cell Reports. 39: 110857 |
Longatti A, Ponzoni L, Moretto E, et al. (2021) Arhgap22 Disruption Leads to RAC1 Hyperactivity Affecting Hippocampal Glutamatergic Synapses and Cognition in Mice. Molecular Neurobiology |
Pizzamiglio L, Focchi E, Cambria CM, et al. (2020) The DNA repair protein ATM as target in autism spectrum disorder. Jci Insight |
Mancini M, Bassani S, Passafaro M. (2020) Right Place at the Right Time: How Changes in Protocadherins Affect Synaptic Connections Contributing to the Etiology of Neurodevelopmental Disorders. Cells. 9 |
Regoni M, Cattaneo S, Mercatelli D, et al. (2020) Pharmacological antagonism of kainate receptor rescues dysfunction and loss of dopamine neurons in a mouse model of human parkin-induced toxicity. Cell Death & Disease. 11: 963 |