Bulent Ataman, ph.D.

Affiliations: 
Neurobiology Harvard Medical School, Boston, MA, United States 
Area:
Activity-dependent regulation of neurons and synapses
Website:
http://connects.catalyst.harvard.edu/profiles/profile/person/37824
Google:
"Bulent Ataman"
Mean distance: 14.28 (cluster 11)
 
SNBCP
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Publications

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Schmitz-Abe K, Sanchez-Schmitz G, Doan RN, et al. (2020) Homozygous deletions implicate non-coding epigenetic marks in Autism spectrum disorder. Scientific Reports. 10: 14045
Ataman B, Boulting GL, Harmin DA, et al. (2016) Evolution of Osteocrin as an activity-regulated factor in the primate brain. Nature. 539: 242-247
Loebrich S, Rathje M, Hager E, et al. (2015) Genomic Mapping and Cellular Expression of HumanCPG2Transcripts in theSYNE1Gene. Molecular and Cellular Neurosciences
Yu TW, Chahrour MH, Coulter ME, et al. (2013) Using whole-exome sequencing to identify inherited causes of autism. Neuron. 77: 259-73
Soskis MJ, Ho HY, Bloodgood BL, et al. (2012) A chemical genetic approach reveals distinct EphB signaling mechanisms during brain development. Nature Neuroscience. 15: 1645-54
Speese SD, Ashley J, Jokhi V, et al. (2012) Nuclear envelope budding enables large ribonucleoprotein particle export during synaptic Wnt signaling. Cell. 149: 832-46
Chahrour MH, Yu TW, Lim ET, et al. (2012) Whole-exome sequencing and homozygosity analysis implicate depolarization-regulated neuronal genes in autism. Plos Genetics. 8: e1002635
Korkut C, Ataman B, Ramachandran P, et al. (2009) Trans-synaptic transmission of vesicular Wnt signals through Evi/Wntless. Cell. 139: 393-404
Fuentes-Medel Y, Logan MA, Ashley J, et al. (2009) Glia and muscle sculpt neuromuscular arbors by engulfing destabilized synaptic boutons and shed presynaptic debris. Plos Biology. 7: e1000184
Ataman B, Ashley J, Gorczyca M, et al. (2008) Rapid activity-dependent modifications in synaptic structure and function require bidirectional Wnt signaling. Neuron. 57: 705-18
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