Bulent Ataman, ph.D.
Affiliations: | Neurobiology | Harvard Medical School, Boston, MA, United States |
Area:
Activity-dependent regulation of neurons and synapsesWebsite:
http://connects.catalyst.harvard.edu/profiles/profile/person/37824Google:
"Bulent Ataman"Mean distance: 14.28 (cluster 11) | S | N | B | C | P |
Parents
Sign in to add mentor| Vivian Budnik | grad student | 2003-2008 | UMass Med School |
| Michael E. Greenberg | post-doc | 2009- | Harvard Medical School |
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Publications
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| Boulting GL, Durresi E, Ataman B, et al. (2021) Activity-dependent regulome of human GABAergic neurons reveals new patterns of gene regulation and neurological disease heritability. Nature Neuroscience |
| Schmitz-Abe K, Sanchez-Schmitz G, Doan RN, et al. (2020) Homozygous deletions implicate non-coding epigenetic marks in Autism spectrum disorder. Scientific Reports. 10: 14045 |
| Ataman B, Boulting GL, Harmin DA, et al. (2016) Evolution of Osteocrin as an activity-regulated factor in the primate brain. Nature. 539: 242-247 |
| Loebrich S, Rathje M, Hager E, et al. (2015) Genomic Mapping and Cellular Expression of HumanCPG2Transcripts in theSYNE1Gene. Molecular and Cellular Neurosciences |
| Yu TW, Chahrour MH, Coulter ME, et al. (2013) Using whole-exome sequencing to identify inherited causes of autism. Neuron. 77: 259-73 |
| Soskis MJ, Ho HY, Bloodgood BL, et al. (2012) A chemical genetic approach reveals distinct EphB signaling mechanisms during brain development. Nature Neuroscience. 15: 1645-54 |
| Speese SD, Ashley J, Jokhi V, et al. (2012) Nuclear envelope budding enables large ribonucleoprotein particle export during synaptic Wnt signaling. Cell. 149: 832-46 |
| Chahrour MH, Yu TW, Lim ET, et al. (2012) Whole-exome sequencing and homozygosity analysis implicate depolarization-regulated neuronal genes in autism. Plos Genetics. 8: e1002635 |
| Korkut C, Ataman B, Ramachandran P, et al. (2009) Trans-synaptic transmission of vesicular Wnt signals through Evi/Wntless. Cell. 139: 393-404 |
| Fuentes-Medel Y, Logan MA, Ashley J, et al. (2009) Glia and muscle sculpt neuromuscular arbors by engulfing destabilized synaptic boutons and shed presynaptic debris. Plos Biology. 7: e1000184 |