Carlo L. Condello, Ph.D.
Affiliations: | Institute for Neurodegenerative Diseases | University of California, San Francisco, San Francisco, CA |
Area:
Neurodegenerative diseases, Biology of Neuron-Glial interactionsGoogle:
"Carlo Condello"Mean distance: 15.5 (cluster 31) | S | N | B | C | P |
Cross-listing: Neuropathology Tree
Parents
Sign in to add mentor| Jaime Grutzendler | grad student | 2004-2011 | UCSF School of Medicine | |
| (T) | ||||
| Stanley Ben Prusiner | post-doc | 2012- | UCSF | |
BETA: Related publications
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Publications
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| Condello C, Merz GE, Aoyagi A, et al. (2022) Aβ and Tau Prions Causing Alzheimer's Disease. Methods in Molecular Biology (Clifton, N.J.). 2561: 293-337 |
| Condello C, Maxwell AM, Castillo E, et al. (2022) Aβ and tau prions feature in the neuropathogenesis of Down syndrome. Proceedings of the National Academy of Sciences of the United States of America. 119: e2212954119 |
| Wang C, Fan L, Khawaja RR, et al. (2022) Microglial NF-κB drives tau spreading and toxicity in a mouse model of tauopathy. Nature Communications. 13: 1969 |
| Ayers JI, Lee J, Monteiro O, et al. (2022) Different α-synuclein prion strains cause dementia with Lewy bodies and multiple system atrophy. Proceedings of the National Academy of Sciences of the United States of America. 119 |
| Belsare KD, Wu H, Mondal D, et al. (2022) Soluble TREM2 inhibits secondary nucleation of Aβ fibrillization and enhances cellular uptake of fibrillar Aβ. Proceedings of the National Academy of Sciences of the United States of America. 119 |
| Maxwell AM, Yuan P, Rivera BM, et al. (2021) Emergence of distinct and heterogeneous strains of amyloid beta with advanced Alzheimer's disease pathology in Down syndrome. Acta Neuropathologica Communications. 9: 201 |
| Condello C, DeGrado WF, Prusiner SB. (2020) Prion biology: implications for Alzheimer's disease therapeutics. The Lancet. Neurology. 19: 802-803 |
| Aoyagi A, Condello C, Stöhr J, et al. (2019) Aβ and tau prion-like activities decline with longevity in the Alzheimer's disease human brain. Science Translational Medicine. 11 |
| Didonna A, Cantó E, Shams H, et al. (2019) Sex-specific Tau methylation patterns and synaptic transcriptional alterations are associated with neural vulnerability during chronic neuroinflammation. Journal of Autoimmunity |
| Mok SA, Condello C, Freilich R, et al. (2018) Mapping interactions with the chaperone network reveals factors that protect against tau aggregation. Nature Structural & Molecular Biology. 25: 384-393 |