Velia M. Fowler
Affiliations: | Scripps Research Institute, La Jolla, La Jolla, CA, United States |
Area:
Cell Biology, General BiophysicsGoogle:
"Velia Fowler"Mean distance: (not calculated yet)
Children
Sign in to add trainee| Andria Lee | grad student | 2000 | Scripps Institute |
| Ryan S. Littlefield | grad student | 2000 | Scripps Institute |
| Carol C. Gregorio | post-doc | Scripps Institute (Anatomy Tree) | |
| Philip A. Kuhlman | post-doc | 1995 | Scripps Institute |
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Publications
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| Vasilescu C, Colpan M, Ojala TH, et al. (2024) Recessive TMOD1 mutation causes childhood cardiomyopathy. Communications Biology. 7: 7 |
| Ghosh A, Coffin M, West R, et al. (2022) Erythroid differentiation in mouse erythroleukemia cells depends on Tmod3-mediated regulation of actin filament assembly into the erythroblast membrane skeleton. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 36: e22220 |
| Nowak RB, Alimohamadi H, Pestonjamasp K, et al. (2022) Nanoscale Dynamics of Actin Filaments in the Red Blood Cell Membrane Skeleton. Molecular Biology of the Cell. mbcE21030107 |
| Ghosh A, Fowler VM. (2021) Tropomodulins. Current Biology : Cb. 31: R501-R503 |
| Parreno J, Amadeo MB, Kwon EH, et al. (2020) Tropomyosin 3.1 Association With Actin Stress Fibers is Required for Lens Epithelial to Mesenchymal Transition. Investigative Ophthalmology & Visual Science. 61: 2 |
| Alimohamadi H, Smith AS, Nowak RB, et al. (2020) Non-uniform distribution of myosin-mediated forces governs red blood cell membrane curvature through tension modulation. Plos Computational Biology. 16: e1007890 |
| Barnum CE, Al Saai S, Patel SD, et al. (2020) The Tudor-domain protein TDRD7, mutated in congenital cataract, controls the heat shock protein HSPB1 (HSP27) and lens fiber cell morphology. Human Molecular Genetics |
| Pal K, Nowak R, Billington N, et al. (2020) Megakaryocyte migration defects due to nonmuscle myosin IIA mutations underly thrombocytopenia in MYH9-Related Disease. Blood |
| Alimohamadi H, Smith A, Fowler V, et al. (2020) Red Blood Cell Curvature is Controlled by the Non-uniform Distribution of Myosin-Mediated Forces and Membrane Tension Biophysical Journal. 118: 231a |
| Smith AS, Pal K, Nowak RB, et al. (2019) MYH9-related disease mutations cause abnormal red blood cell morphology through increased myosin-actin binding at the membrane. American Journal of Hematology |