Marina N. Asiedu

University of Arizona, Tucson, AZ 
"Marina Asiedu"
Mean distance: 17.86 (cluster 32)


Sign in to add mentor
Theodore J. Price grad student 2012 University of Arizona
 (Spinal sensitization mechanisms promoting pain: GABAergic disinhibition and PKMzeta-mediated plasticity.)
BETA: Related publications


You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect.

Moy JK, Szabo-Pardi T, Tillu DV, et al. (2019) Temporal and sex differences in the role of BDNF/TrkB signaling in hyperalgesic priming in mice and rats. Neurobiology of Pain (Cambridge, Mass.). 5: 100024
Moy JK, Khoutorsky A, Asiedu MN, et al. (2018) eIF4E Phosphorylation InfluencesmRNA Translation in Mouse Dorsal Root Ganglion Neurons. Frontiers in Cellular Neuroscience. 12: 29
Burton MD, Tillu DV, Mazhar K, et al. (2017) Pharmacological activation of AMPK inhibits incision-evoked mechanical hypersensitivity and the development of hyperalgesic priming in mice. Neuroscience
Moy JK, Khoutorsky A, Asiedu MN, et al. (2017) The MNK - eIF4E signaling axis contributes to injury-induced nociceptive plasticity and the development of chronic pain. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience
Asiedu MN, Han C, Dib-Hajj SD, et al. (2017) The AMPK Activator A769662 Blocks Voltage-Gated Sodium Channels: Discovery of a Novel Pharmacophore with Potential Utility for Analgesic Development. Plos One. 12: e0169882
Asiedu MN, Dussor G, Price TJ. (2016) Targeting AMPK for the Alleviation of Pathological Pain. Exs. 107: 257-285
Mejia GL, Asiedu MN, Hitoshi Y, et al. (2016) The potent, indirect adenosine monophosphate- activated protein kinase activator R419 attenuates mitogen-activated protein kinase signaling, inhibits nociceptor excitability, and reduces pain hypersensitivity in mice. Pain Reports (Baltimore, Md.). 1
Kim JV, Megat S, Moy JK, et al. (2016) Neuroligin-2 regulates spinal GABAergic plasticity in hyperalgesic priming, a model of the transition from acute to chronic pain. Pain
Boitano S, Hoffman J, Flynn AN, et al. (2015) The novel PAR2 ligand C391 blocks multiple PAR2 signaling pathways in vitro and in vivo. British Journal of Pharmacology
Kim JY, Tillu DV, Quinn TL, et al. (2015) Spinal dopaminergic projections control the transition to pathological pain plasticity via a D1/D5-mediated mechanism. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 35: 6307-17
See more...