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Amy E. Juedes, Ph.D.

Affiliations: 
2001 Yale University, New Haven, CT 
Area:
Immunology, Neuroscience Biology, Molecular Biology
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Nancy H. Ruddle grad student 2001 Yale
 (Regulation of antigen presentation and inflammation in the central nervous system during experimental autoimmune encephalomyelitis.)

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Publications

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Fousteri G, Dave A, Juedes A, et al. (2011) Increased memory conversion of naïve CD8 T cells activated during late phases of acute virus infection due to decreased cumulative antigen exposure. Plos One. 6: e14502
Coppieters K, Barral AM, Juedes A, et al. (2009) No significant CTL cross-priming by dendritic cell-derived exosomes during murine lymphocytic choriomeningitis virus infection. Journal of Immunology (Baltimore, Md. : 1950). 182: 2213-20
Filippi CM, Juedes AE, Oldham JE, et al. (2008) Transforming growth factor-beta suppresses the activation of CD8+ T-cells when naive but promotes their survival and function once antigen experienced: a two-faced impact on autoimmunity. Diabetes. 57: 2684-92
Martinic MM, Juedes AE, Bresson D, et al. (2007) Minimal impact of a de novo-expressed beta-cell autoantigen on spontaneous diabetes development in NOD mice. Diabetes. 56: 1059-68
Haase C, Ejrnaes M, Juedes AE, et al. (2005) Immunomodulatory dendritic cells require autologous serum to circumvent nonspecific immunosuppressive activity in vivo. Blood. 106: 4225-33
Juedes AE, Von Herrath MG. (2005) Antigen-specific regulation of autoimmunity Molecular Autoimmunity. 407-417
Juedes AE, von Herrath MG. (2004) Regulatory T-cells in type 1 diabetes. Diabetes/Metabolism Research and Reviews. 20: 446-51
Juedes AE, Rodrigo E, Togher L, et al. (2004) T-bet controls autoaggressive CD8 lymphocyte responses in type 1 diabetes. The Journal of Experimental Medicine. 199: 1153-62
Christen U, Juedes A, Homann D, et al. (2004) Virally induced inflammation and therapeutic avenues in type 1 diabetes. Endocrinology and Metabolism Clinics of North America. 33: 45-58, viii
Juedes AE, Von Herrath MG. (2003) Using regulatory APCs to induce/maintain tolerance. Annals of the New York Academy of Sciences. 1005: 128-37
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