Diane M. Papazian
Affiliations: | University of California, Los Angeles, Los Angeles, CA |
Area:
K ChannelsWebsite:
http://149.142.237.180/faculty/papazian.htmGoogle:
"Diane Papazian"Mean distance: 15.92 (cluster 11)
Children
Sign in to add trainee| William R. Silverman | grad student | 2002 | UCLA |
| Natali A. Minassian | grad student | 2010 | UCLA |
| Naomi Nagaya | post-doc | UCLA | |
| Seema K. Tiwari-Woodruff | post-doc | 1994-1998 | UCLA (Cell Biology Tree) |
| Michael P. Myers | post-doc | 1998-2001 | UCLA |
| Fadi A. Issa | post-doc | 2009-2014 | UCLA |
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Publications
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| Bavan S, Goodkin HP, Papazian DM. (2022) Altered Closed State Inactivation Gating in Kv4.2 Channels Results in Developmental and Epileptic Encephalopathies in Human Patients. Human Mutation |
| Hsieh JY, Ulrich BN, Issa FA, et al. (2020) Infant and adult SCA13 mutations differentially affect Purkinje cell excitability, maturation, and viability in vivo. Elife. 9 |
| Hsieh J, Ulrich BN, Issa FA, et al. (2020) Author response: Infant and adult SCA13 mutations differentially affect Purkinje cell excitability, maturation, and viability in vivo Elife |
| Lin MA, Cannon SC, Papazian DM. (2018) Kv4.2 autism and epilepsy mutation enhances inactivation of closed channels but impairs access to inactivated state after opening. Proceedings of the National Academy of Sciences of the United States of America |
| Hsieh JY, Papazian DM. (2018) In Vivo Analysis of Potassium Channelopathies: Loose Patch Recording of Purkinje Cell Firing in Living, Awake Zebrafish. Methods in Molecular Biology (Clifton, N.J.). 1684: 237-252 |
| Duarri A, Lin MC, Fokkens MR, et al. (2015) Spinocerebellar ataxia type 19/22 mutations alter heterocomplex Kv4.3 channel function and gating in a dominant manner. Cellular and Molecular Life Sciences : Cmls |
| Hsieh JY, Ulrich B, Issa FA, et al. (2014) Rapid development of Purkinje cell excitability, functional cerebellar circuit, and afferent sensory input to cerebellum in zebrafish. Frontiers in Neural Circuits. 8: 147 |
| Lee H, Lin MC, Kornblum HI, et al. (2014) Exome sequencing identifies de novo gain of function missense mutation in KCND2 in identical twins with autism and seizures that slows potassium channel inactivation. Human Molecular Genetics. 23: 3481-9 |
| Lin MA, Lee H, Kornblum HI, et al. (2014) Kcnd2 Mutation Associated with Autism and Epilepsy Impairs Inactivation Gating in Kv4.2 K+ Channels Biophysical Journal. 106: 741a |
| Issa FA, Mock AF, Sagasti A, et al. (2012) Spinocerebellar ataxia type 13 mutation that is associated with disease onset in infancy disrupts axonal pathfinding during neuronal development. Disease Models & Mechanisms. 5: 921-9 |