Victor S. Van Laar, Ph.D.
Affiliations: | 2008 | University of Pittsburgh, Pittsburgh, PA, United States |
Area:
Neuroscience BiologyGoogle:
"Victor Van Laar"Mean distance: 35622
Parents
Sign in to add mentor| Teresa Hastings | grad student | 2008 | University of Pittsburgh | |
| (Proteomic analysis of dopamine oxidation induced modifications to mitochondrial proteins: Implications for Parkinson's disease.) | ||||
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Publications
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| Van Laar AD, Webb KR, Keeney MT, et al. (2023) Transient exposure to rotenone causes degeneration and progressive parkinsonian motor deficits, neuroinflammation, and synucleinopathy. Npj Parkinson's Disease. 9: 121 |
| Van Laar AD, Van Laar VS, San Sebastian W, et al. (2021) An Update on Gene Therapy Approaches for Parkinson's Disease: Restoration of Dopaminergic Function. Journal of Parkinson's Disease |
| Van Laar VS, Chen J, Zharikov AD, et al. (2020) α-Synuclein amplifies cytoplasmic peroxide flux and oxidative stress provoked by mitochondrial inhibitors in CNS dopaminergic neurons in vivo. Redox Biology. 37: 101695 |
| Van Laar VS, Otero PA, Hastings TG, et al. (2018) Potential Role of Mic60/Mitofilin in Parkinson's Disease. Frontiers in Neuroscience. 12: 898 |
| Van Laar VS, Arnold B, Howlett EH, et al. (2018) Evidence for compartmentalized axonal mitochondrial biogenesis: Mitochondrial DNA replication increases in distal axons as an early response to Parkinson's disease-relevant stress. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience |
| Dukes AA, Bai Q, Van Laar VS, et al. (2016) Live imaging of mitochondrial dynamics in CNS dopaminergic neurons in vivo demonstrates early reversal of mitochondrial transport following MPP(+) exposure. Neurobiology of Disease |
| Van Laar VS, Berman SB, Hastings TG. (2016) Mic60/mitofilin overexpression alters mitochondrial dynamics and attenuates vulnerability of dopaminergic cells to dopamine and rotenone. Neurobiology of Disease. 91: 247-61 |
| Van Laar VS, Roy N, Liu A, et al. (2015) Glutamate excitotoxicity in neurons triggers mitochondrial and endoplasmic reticulum accumulation of Parkin, and, in the presence of N-acetyl cysteine, mitophagy. Neurobiology of Disease. 74: 180-93 |
| Van Laar VS, Berman SB. (2013) The interplay of neuronal mitochondrial dynamics and bioenergetics: implications for Parkinson's disease. Neurobiology of Disease. 51: 43-55 |
| Van Laar VS, Arnold B, Cassady SJ, et al. (2011) Bioenergetics of neurons inhibit the translocation response of Parkin following rapid mitochondrial depolarization. Human Molecular Genetics. 20: 927-40 |