Helena Decker
Affiliations: | Oregon Health and Science University, Portland, OR |
Area:
polarity neuronal, axonal transport, AlzheimerGoogle:
"Helena Decker"Mean distance: 18.07 (cluster 58)
Parents
Sign in to add mentor| Sergio T. Ferreira | grad student | 2007-2011 | Federal University of Rio de Janeiro |
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Publications
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| Ramser EM, Gan KJ, Decker H, et al. (2013) Amyloid-β oligomers induce tau-independent disruption of BDNF axonal transport via calcineurin activation in cultured hippocampal neurons. Molecular Biology of the Cell. 24: 2494-505 |
| Bomfim TR, Forny-Germano L, Sathler LB, et al. (2012) An anti-diabetes agent protects the mouse brain from defective insulin signaling caused by Alzheimer's disease- associated Aβ oligomers. The Journal of Clinical Investigation. 122: 1339-53 |
| Decker H, Jürgensen S, Adrover MF, et al. (2010) N-methyl-D-aspartate receptors are required for synaptic targeting of Alzheimer's toxic amyloid-β peptide oligomers. Journal of Neurochemistry. 115: 1520-9 |
| Decker H, Lo KY, Unger SM, et al. (2010) Amyloid-beta peptide oligomers disrupt axonal transport through an NMDA receptor-dependent mechanism that is mediated by glycogen synthase kinase 3beta in primary cultured hippocampal neurons. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 30: 9166-71 |
| De Felice FG, Vieira MN, Bomfim TR, et al. (2009) Protection of synapses against Alzheimer's-linked toxins: insulin signaling prevents the pathogenic binding of Abeta oligomers. Proceedings of the National Academy of Sciences of the United States of America. 106: 1971-6 |
| Felice FGD, Vieira MNN, Bomfim TR, et al. (2009) Erratum: Protection of synapses against Alzheimer's- Linked toxins: Insulin signaling prevents the pathogenic binding of Aβ oligomers (Proceedings of the National Academy of Sciences of the United States of America (2009) 106 (1971-1976) DOI:10.1073/pnas.0809158106) Proceedings of the National Academy of Sciences of the United States of America. 106 |