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Toma C, Pierce KD, Shaw AD, et al. (2018) Comprehensive cross-disorder analyses of CNTNAP2 suggest it is unlikely to be a primary risk gene for psychiatric disorders. Plos Genetics. 14: e1007535
Toma C, Shaw AD, Allcock RJN, et al. (2018) An examination of multiple classes of rare variants in extended families with bipolar disorder. Translational Psychiatry. 8: 65
Salminen LE, Schofield PR, Pierce KD, et al. (2017) Vulnerability of white matter tracts and cognition to the SOD2 polymorphism: A preliminary study of antioxidant defense genes in brain aging. Behavioural Brain Research
Routledge KM, Burton KL, Williams LM, et al. (2016) The shared and unique genetic relationship between mental well-being, depression and anxiety symptoms and cognitive function in healthy twins. Cognition & Emotion. 1-15
Meiser B, Peate M, Levitan C, et al. (2016) A Psycho-Educational Intervention for People with a Family History of Depression: Pilot Results. Journal of Genetic Counseling
Lim YY, Hassenstab J, Cruchaga C, et al. (2016) BDNF Val66Met moderates memory impairment, hippocampal function and tau in preclinical autosomal dominant Alzheimer's disease. Brain : a Journal of Neurology
Routledge KM, Burton KL, Williams LM, et al. (2016) Shared versus distinct genetic contributions of mental wellbeing with depression and anxiety symptoms in healthy twins. Psychiatry Research. 244: 65-70
Shiner CT, Pierce KD, Thompson-Butel AG, et al. (2016) BDNF Genotype Interacts with Motor Function to Influence Rehabilitation Responsiveness Poststroke. Frontiers in Neurology. 7: 69
Lee S, Viqar F, Zimmerman ME, et al. (2016) White matter hyperintensities are a core feature of Alzheimer's disease: Evidence from the Dominantly Inherited Alzheimer Network. Annals of Neurology
Su Y, Blazey TM, Owen CJ, et al. (2016) Quantitative Amyloid Imaging in Autosomal Dominant Alzheimer's Disease: Results from the DIAN Study Group. Plos One. 11: e0152082
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