Graham V. Goddard

Affiliations: 
Dalhousie University, Halifax, Nova Scotia, Canada 
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"Graham Goddard"
Mean distance: 12.71 (cluster 17)
 
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Publications

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Holmes KH, Bilkey DK, Laverty R, et al. (1990) The N-methyl-D-aspartate antagonists aminophosphonovalerate and carboxypiperazinephosphonate retard the development and expression of kindled seizures. Brain Research. 506: 227-35
Otani S, Marshall CJ, Tate WP, et al. (1989) Maintenance of long-term potentiation in rat dentate gyrus requires protein synthesis but not messenger RNA synthesis immediately post-tetanization. Neuroscience. 28: 519-26
Morimoto K, Goddard GV. (1988) Seizure-triggering mechanism in the kindling model of epilepsy: I. EEG changes during stimulation from the site of stimulation. The Japanese Journal of Psychiatry and Neurology. 42: 618-9
Morimoto K, Goddard GV. (1987) The substantia nigra is an important site for the containment of seizure generalization in the kindling model of epilepsy. Epilepsia. 28: 1-10
Morimoto K, Mason SE, Goddard GV. (1987) Kindling-induced changes in the EEG recorded during stimulation from the site of stimulation. II. Comparison between spontaneous and evoked potentials. Experimental Neurology. 97: 1-16
Dragunow M, Goddard GV, Laverty R. (1987) Proconvulsant effects of theophylline on hippocampal afterdischarges. Experimental Neurology. 96: 732-5
Morimoto K, Otani S, Goddard GV. (1987) Effects of acute and long-term treatment with amphetamine on evoked responses and long-term potentiation in the dentate gyrus of anesthetized rats. Brain Research. 407: 137-43
Bilkey DK, Goddard GV. (1987) Septohippocampal and commissural pathways antagonistically control inhibitory interneurons in the dentate gyrus. Brain Research. 405: 320-5
Maru E, Goddard GV. (1987) Alteration in dentate neuronal activities associated with perforant path kindling. III. Enhancement of synaptic inhibition. Experimental Neurology. 96: 46-60
Maru E, Goddard GV. (1987) Alteration in dentate neuronal activities associated with perforant path kindling. II. Decrease in granule cell excitability. Experimental Neurology. 96: 33-45
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