Mizuki Yamada

Laboratory for Structural Neuropathology RIKEN Brain Science Institute, Wakō-shi, Saitama-ken, Japan 
"Mizuki Yamada"
Mean distance: 106866 (cluster 31)
BETA: Related publications


You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect.

Kino Y, Washizu C, Kurosawa M, et al. (2016) FUS/TLS acts as an aggregation-dependent modifier of polyglutamine disease model mice. Scientific Reports. 6: 35236
Kino Y, Washizu C, Kurosawa M, et al. (2015) FUS/TLS deficiency causes behavioral and pathological abnormalities distinct from amyotrophic lateral sclerosis. Acta Neuropathologica Communications. 3: 24
Kino Y, Washizu C, Aquilanti E, et al. (2011) Intracellular localization and splicing regulation of FUS/TLS are variably affected by amyotrophic lateral sclerosis-linked mutations. Nucleic Acids Research. 39: 2781-98
Bauer PO, Goswami A, Wong HK, et al. (2010) Harnessing chaperone-mediated autophagy for the selective degradation of mutant huntingtin protein. Nature Biotechnology. 28: 256-63
Yamanaka T, Tosaki A, Miyazaki H, et al. (2010) Mutant huntingtin fragment selectively suppresses Brn-2 POU domain transcription factor to mediate hypothalamic cell dysfunction. Human Molecular Genetics. 19: 2099-112
Miyazaki H, Oyama F, Kurosawa M, et al. (2009) Correlation between nuclear accumulation and dysregulation of sodium channel β4 subunit in HD transgenic mice Neuroscience Research. 65: S247
Wong HK, Bauer PO, Kurosawa M, et al. (2008) Blocking acid-sensing ion channel 1 alleviates Huntington's disease pathology via an ubiquitin-proteasome system-dependent mechanism. Human Molecular Genetics. 17: 3223-35
Nucifora FC, Sasaki M, Peters MF, et al. (2001) Interference by huntingtin and atrophin-1 with cbp-mediated transcription leading to cellular toxicity. Science (New York, N.Y.). 291: 2423-8
See more...