Jesse E. Hanson
Affiliations: | Stanford University School of Medicine, Palo Alto, CA, United States |
Area:
Synaptic Physiology, Fragile X, ParkinsonsGoogle:
"Jesse Hanson"Mean distance: 13.45 (cluster 6) | S | N | B | C | P |
Parents
Sign in to add mentor| Dieter Jaeger | grad student | 2003 | Emory | |
| (Synaptic integration of excitatory inputs in globus pallidus neurons.) | ||||
| Yoland Smith | grad student | 2003 | Emory | |
| (Synaptic integration of excitatory inputs in globus pallidus neurons.) | ||||
| Daniel V. Madison | post-doc | Stanford | ||
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Publications
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| Dejanovic B, Sheng M, Hanson JE. (2023) Targeting synapse function and loss for treatment of neurodegenerative diseases. Nature Reviews. Drug Discovery |
| Hanson JE, Yuan H, Perszyk RE, et al. (2023) Therapeutic potential of N-methyl-D-aspartate receptor modulators in psychiatry. Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology |
| Zhou J, Wade SD, Graykowski D, et al. (2023) The neuronal pentraxin Nptx2 regulates complement activity and restrains microglia-mediated synapse loss in neurodegeneration. Science Translational Medicine. 15: eadf0141 |
| Dejanovic B, Wu T, Tsai MC, et al. (2022) Complement C1q-dependent excitatory and inhibitory synapse elimination by astrocytes and microglia in Alzheimer's disease mouse models. Nature Aging. 2: 837-850 |
| Wang Y, Kerrisk Campbell M, Tom I, et al. (2020) PCDH7 interacts with GluN1 and regulates dendritic spine morphology and synaptic function. Scientific Reports. 10: 10951 |
| Hanson JE, Ma K, Elstrott J, et al. (2020) GluN2A NMDA Receptor Enhancement Improves Brain Oscillations, Synchrony, and Cognitive Functions in Dravet Syndrome and Alzheimer's Disease Models. Cell Reports. 30: 381-396.e4 |
| Huntley MA, Srinivasan K, Friedman BA, et al. (2019) Genome-wide analysis of differential gene expression and splicing in excitatory neurons and interneuron subtypes. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience |
| Wu T, Dejanovic B, Gandham VD, et al. (2019) Complement C3 Is Activated in Human AD Brain and Is Required for Neurodegeneration in Mouse Models of Amyloidosis and Tauopathy. Cell Reports. 28: 2111-2123.e6 |
| Yao L, Grand T, Hanson JE, et al. (2018) Author Correction: Higher ambient synaptic glutamate at inhibitory versus excitatory neurons differentially impacts NMDA receptor activity. Nature Communications. 9: 4887 |
| Dejanovic B, Huntley MA, De Mazière A, et al. (2018) Changes in the Synaptic Proteome in Tauopathy and Rescue of Tau-Induced Synapse Loss by C1q Antibodies. Neuron |