Michael Paul Murphy

University of Kentucky University of Kentucky, Lexington, KY 
Alzheimer's disease
"Michael Murphy"
Mean distance: 17.5 (cluster 28)
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Kim JH, Choi TG, Park S, et al. (2018) Mitochondrial ROS-derived PTEN oxidation activates PI3K pathway for mTOR-induced myogenic autophagy. Cell Death and Differentiation
Xi Y, Feng D, Tao K, et al. (2018) MitoQ protects dopaminergic neurons in a 6-OHDA induced PD model by enhancing Mfn2-dependent mitochondrial fusion via activation of PGC-1α. Biochimica Et Biophysica Acta
Platt TL, Beckett TL, Kohler K, et al. (2016) Obesity, diabetes, and leptin resistance promote tau pathology in a mouse model of disease Neuroscience. 315: 162-174
Wilcock DM, Hurban J, Helman AM, et al. (2015) Down syndrome individuals with Alzheimer's disease have a distinct neuroinflammatory phenotype compared to sporadic Alzheimer's disease. Neurobiology of Aging. 36: 2468-74
Tramutola A, Triplett JC, Di Domenico F, et al. (2015) Alteration of mTOR signaling occurs early in the progression of Alzheimer disease (AD): analysis of brain from subjects with pre-clinical AD, amnestic mild cognitive impairment and late-stage AD. Journal of Neurochemistry. 133: 739-49
Sethi M, Joshi SS, Webb RL, et al. (2015) Increased fragmentation of sleep-wake cycles in the 5XFAD mouse model of Alzheimer's disease. Neuroscience. 290: 80-9
Helman AM, Murphy MP. (2015) Vascular cognitive impairment: Modeling a critical neurologic disease in vitro and in vivo Biochimica Et Biophysica Acta - Molecular Basis of Disease
Holler CJ, Davis PR, Beckett TL, et al. (2014) Bridging integrator 1 (BIN1) protein expression increases in the Alzheimer's disease brain and correlates with neurofibrillary tangle pathology. Journal of Alzheimer's Disease : Jad. 42: 1221-7
Martin SB, Dowling AL, Lianekhammy J, et al. (2014) Synaptophysin and synaptojanin-1 in Down syndrome are differentially affected by Alzheimer's disease. Journal of Alzheimer's Disease : Jad. 42: 767-75
Niedowicz DM, Reeves VL, Platt TL, et al. (2014) Obesity and diabetes cause cognitive dysfunction in the absence of accelerated β-amyloid deposition in a novel murine model of mixed or vascular dementia. Acta Neuropathologica Communications. 2: 64
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