Feng Yang, Ph.D
Affiliations: | Lieber Institute for Brain Development |
Area:
Neuronal Development and Synaptic PlasticityWebsite:
http://www.libd.orgGoogle:
"Feng Yang"Mean distance: 13.51 (cluster 6) | S | N | B | C | P |
Parents
Sign in to add mentorChien-Ping Wu | grad student | ION, CAS | |
Bai Lu | post-doc | NICHD | |
Daniel R. Weinberger | research scientist | Lieber Institute for Brain Development |
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Publications
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Mei F, Zhao C, Li S, et al. (2024) Ngfr cholinergic projection from SI/nBM to mPFC selectively regulates temporal order recognition memory. Nature Communications. 15: 7342 |
Yang F, You H, Mizui T, et al. (2024) Inhibiting proBDNF to mature BDNF conversion leads to ASD-like phenotypes in vivo. Molecular Psychiatry |
Zhang T, Li S, Mei F, et al. (2022) A protocol for establishing a male G×E schizophrenia mouse model. Star Protocols. 3: 101856 |
Chen Y, Li S, Zhang T, et al. (2022) Corticosterone antagonist or TrkB agonist attenuates schizophrenia-like behavior in a mouse model combining deficiency and developmental stress. Iscience. 25: 104609 |
Lan J, Hu Y, Wang X, et al. (2021) Abnormal spatiotemporal expression pattern of progranulin and neurodevelopment impairment in VPA-induced ASD rat model. Neuropharmacology. 108689 |
Lu B, Zhang T, Yang F. (2021) "Bone" in the Brain? Osteocalcin-Expressing Neurons in Adult Hippocampus Promote Neurogenesis and Suppress Anxiety. Biological Psychiatry. 89: 539-540 |
Ren M, Hu Z, Chen Q, et al. (2019) KCNH2-3.1 mediates aberrant complement activation and impaired hippocampal-medial prefrontal circuitry associated with working memory deficits. Molecular Psychiatry |
Hill JL, Jimenez DV, Mai Y, et al. (2018) Cortistatin-expressing interneurons require TrkB signaling to suppress neural hyper-excitability. Brain Structure & Function |
Papaleo F, Yang F, Paterson C, et al. (2016) Behavioral, Neurophysiological, and Synaptic Impairment in a Transgenic Neuregulin1 (NRG1-IV) Murine Schizophrenia Model. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 36: 4859-75 |
Carr GV, Chen J, Yang F, et al. (2016) KCNH2-3.1 expression impairs cognition and alters neuronal function in a model of molecular pathology associated with schizophrenia. Molecular Psychiatry |