Mahmoud L. Soliman, Ph.D.

Affiliations: 
2012 Pharmacology, Physiology & Therapeutics The University of North Dakota 
Area:
Pharmacology, Neuroscience Biology
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Thad A. Rosenberger grad student 2012 The University of North Dakota
 (Neuroglial mechanisms involved in the anti-inflammatory effect of acetate supplementation.)
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Publications

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Li S, Geiger NH, Soliman ML, et al. (2015) Caffeine, Through Adenosine A3 Receptor-Mediated Actions, Suppresses Amyloid-β Protein Precursor Internalization and Amyloid-β Generation. Journal of Alzheimer's Disease : Jad. 47: 73-83
Soliman ML, Ohm JE, Rosenberger TA. (2013) Acetate reduces PGE2 release and modulates phospholipase and cyclooxygenase levels in neuroglia stimulated with lipopolysaccharide. Lipids. 48: 651-62
Soliman ML, Combs CK, Rosenberger TA. (2013) Modulation of inflammatory cytokines and mitogen-activated protein kinases by acetate in primary astrocytes. Journal of Neuroimmune Pharmacology : the Official Journal of the Society On Neuroimmune Pharmacology. 8: 287-300
Soliman ML, Puig KL, Combs CK, et al. (2012) Acetate reduces microglia inflammatory signaling in vitro. Journal of Neurochemistry. 123: 555-67
Soliman ML, Smith MD, Houdek HM, et al. (2012) Acetate supplementation modulates brain histone acetylation and decreases interleukin-1β expression in a rat model of neuroinflammation. Journal of Neuroinflammation. 9: 51
Soliman ML, Rosenberger TA. (2011) Acetate supplementation increases brain histone acetylation and inhibits histone deacetylase activity and expression. Molecular and Cellular Biochemistry. 352: 173-80
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