Nathan K. LeBrasseur, Ph.D.

Affiliations: 
2003 Boston University, Boston, MA, United States 
Area:
Cell Biology, Animal Physiology Biology, Recreation
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"Nathan LeBrasseur"
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Cross-listing: Kinesiology Tree

Parents

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Roger A. Fielding grad student 2003 Boston University
 (Regulation of neuregulin/ErbB signaling in skeletal muscle.)
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Publications

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Aversa Z, Zhang X, Fielding RA, et al. (2019) The clinical impact and biological mechanisms of skeletal muscle aging. Bone
Tomita K, Freeman BL, Bronk SF, et al. (2016) CXCL10-Mediates Macrophage, but not Other Innate Immune Cells-Associated Inflammation in Murine Nonalcoholic Steatohepatitis. Scientific Reports. 6: 28786
Schafer MJ, Atkinson EJ, Vanderboom PM, et al. (2016) Quantification of GDF11 and Myostatin in Human Aging and Cardiovascular Disease. Cell Metabolism. 23: 1207-1215
Schafer MJ, White TA, Evans G, et al. (2016) Exercise Prevents Diet-induced Cellular Senescence in Adipose Tissue. Diabetes
Xu M, Tchkonia T, Ding H, et al. (2015) JAK inhibition alleviates the cellular senescence-associated secretory phenotype and frailty in old age. Proceedings of the National Academy of Sciences of the United States of America. 112: E6301-E6310
Bergen HR, Farr JN, Vanderboom PM, et al. (2015) Myostatin as a mediator of sarcopenia versus homeostatic regulator of muscle mass: insights using a new mass spectrometry-based assay. Skeletal Muscle. 5: 21
Zhu Y, Tchkonia T, Pirtskhalava T, et al. (2015) The Achilles' heel of senescent cells: from transcriptome to senolytic drugs. Aging Cell. 14: 644-58
McGee-Lawrence ME, White TA, LeBrasseur NK, et al. (2015) Conditional deletion of Hdac3 in osteoprogenitor cells attenuates diet-induced systemic metabolic dysfunction. Molecular and Cellular Endocrinology. 410: 42-51
Eby SF, Cloud BA, Brandenburg JE, et al. (2015) Shear wave elastography of passive skeletal muscle stiffness: influences of sex and age throughout adulthood. Clinical Biomechanics (Bristol, Avon). 30: 22-7
Idrissova L, Malhi H, Werneburg NW, et al. (2015) TRAIL receptor deletion in mice suppresses the inflammation of nutrient excess. Journal of Hepatology. 62: 1156-63
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