Michael Sereda, M.D.

Affiliations: 
Clinical Neurophysiology Georg-August-Universität Göttingen, Göttingen, Niedersachsen, Germany 
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"Michael Sereda"
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Publications

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Joseph S, Vingill S, Jahn O, et al. (2019) Myelinating glia-specific deletion of Fbxo7 in mice triggers axonal degeneration in the central nervous system together with peripheral neuropathy. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience
Fledrich R, Akkermann D, Schütza V, et al. (2019) Publisher Correction: NRG1 type I dependent autoparacrine stimulation of Schwann cells in onion bulbs of peripheral neuropathies. Nature Communications. 10: 1840
Fledrich R, Akkermann D, Schütza V, et al. (2019) NRG1 type I dependent autoparacrine stimulation of Schwann cells in onion bulbs of peripheral neuropathies. Nature Communications. 10: 1467
Prukop T, Stenzel J, Wernick S, et al. (2019) Early short-term PXT3003 combinational therapy delays disease onset in a transgenic rat model of Charcot-Marie-Tooth disease 1A (CMT1A). Plos One. 14: e0209752
Fledrich R, Mannil M, Leha A, et al. (2017) Biomarkers predict outcome in Charcot- Marie-Tooth disease 1A. Journal of Neurology, Neurosurgery, and Psychiatry
Kleinecke S, Richert S, de Hoz L, et al. (2017) Peroxisomal dysfunctions cause lysosomal storage and axonal Kv1 channel redistribution in peripheral neuropathy. Elife. 6
Günther R, Balck A, Koch JC, et al. (2017) Rho Kinase Inhibition with Fasudil in the SOD1(G93A) Mouse Model of Amyotrophic Lateral Sclerosis-Symptomatic Treatment Potential after Disease Onset. Frontiers in Pharmacology. 8: 17
Sociali G, Visigalli D, Prukop T, et al. (2016) Tolerability and efficacy study of P2X7 inhibition in experimental Charcot-Marie-Tooth type 1A (CMT1A) neuropathy. Neurobiology of Disease
Quintes S, Brinkmann BG, Ebert M, et al. (2016) Zeb2 is essential for Schwann cell differentiation, myelination and nerve repair. Nature Neuroscience
Patzig J, Kusch K, Fledrich R, et al. (2015) Proteolipid protein modulates preservation of peripheral axons and premature death when myelin protein zero is lacking. Glia
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