Ester Feldmesser, Ph.D.
Affiliations: | 2006 | Weizmann Institute of Science, Rehovot, Israel |
Area:
Molecular BiologyGoogle:
"Ester Feldmesser"Mean distance: (not calculated yet)
Parents
Sign in to add mentor| Doron Lancet | grad student | 2006 | Weizmann Institute | |
| (Studies on genes involved in olfaction.) | ||||
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Publications
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| Tsamir-Rimon M, Ben-Dor S, Feldmesser E, et al. (2020) Rapid starch degradation in the wood of olive trees under heat and drought is permitted by three stress-specific beta amylases. The New Phytologist |
| Kohen R, Barlev J, Hornung G, et al. (2019) UTAP: User-friendly Transcriptome Analysis Pipeline. Bmc Bioinformatics. 20: 154 |
| Yoffe Y, David M, Kalaora R, et al. (2016) Cap-independent translation by DAP5 controls cell fate decisions in human embryonic stem cells. Genes & Development. 30: 1991-2004 |
| Leshkowitz D, Feldmesser E, Friedlander G, et al. (2016) Using Synthetic Mouse Spike-In Transcripts to Evaluate RNA-Seq Analysis Tools. Plos One. 11: e0153782 |
| Umansky KB, Feldmesser E, Groner Y. (2015) Genomic-wide transcriptional profiling in primary myoblasts reveals Runx1-regulated genes in muscle regeneration. Genomics Data. 6: 120-2 |
| Pencovich N, Haim O, Feldmesser E, et al. (2015) ATCT-26TUMOR TREATING FIELDS-MEDIATED GENE EXPRESSION IN PATIENTS WITH GBM Neuro-Oncology. 17: v7.2-v7 |
| Feldmesser E, Rosenwasser S, Vardi A, et al. (2014) Improving transcriptome construction in non-model organisms: integrating manual and automated gene definition in Emiliania huxleyi. Bmc Genomics. 15: 148 |
| Keydar I, Ben-Asher E, Feldmesser E, et al. (2013) General olfactory sensitivity database (GOSdb): candidate genes and their genomic variations. Human Mutation. 34: 32-41 |
| Feldmesser E, Rosenwasser S, Vardi A, et al. (2013) Improving automated de-novo transcriptome definition in non-model organisms by integrating manually defined gene information Embnet.Journal. 19: 45 |
| Landau G, Ran A, Bercovich Z, et al. (2012) Expression profiling and biochemical analysis suggest stress response as a potential mechanism inhibiting proliferation of polyamine-depleted cells. The Journal of Biological Chemistry. 287: 35825-37 |