Katherine Kopeikina, PhD

Boston University School of Medicine, Boston, MA 
"Katherine Kopeikina"
Mean distance: 15.82 (cluster 17)
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Varea O, Martin-de-Saavedra MD, Kopeikina KJ, et al. (2015) Synaptic abnormalities and cytoplasmic glutamate receptor aggregates in contactin associated protein-like 2/Caspr2 knockout neurons. Proceedings of the National Academy of Sciences of the United States of America. 112: 6176-81
Smith KR, Kopeikina KJ, Fawcett-Patel JM, et al. (2014) Psychiatric risk factor ANK3/ankyrin-G nanodomains regulate the structure and function of glutamatergic synapses. Neuron. 84: 399-415
Kuchibhotla KV, Wegmann S, Kopeikina KJ, et al. (2014) Neurofibrillary tangle-bearing neurons are functionally integrated in cortical circuits in vivo. Proceedings of the National Academy of Sciences of the United States of America. 111: 510-4
Kopeikina KJ, Wegmann S, Pitstick R, et al. (2013) Tau causes synapse loss without disrupting calcium homeostasis in the rTg4510 model of tauopathy. Plos One. 8: e80834
Kay KR, Smith C, Wright AK, et al. (2013) Studying synapses in human brain with array tomography and electron microscopy. Nature Protocols. 8: 1366-80
Kopeikina KJ, Polydoro M, Tai HC, et al. (2013) Synaptic alterations in the rTg4510 mouse model of tauopathy. The Journal of Comparative Neurology. 521: 1334-53
Kopeikina K. (2013) Morphological and functional consequences of pathological changes in Tau: Are aggregated or soluble forms of tau detrimental to neurons? Rejuvenation Research. 16: 248-249
Kopeikina KJ, Hyman BT, Spires-Jones TL. (2012) Soluble forms of tau are toxic in Alzheimer's disease. Translational Neuroscience. 3: 223-233
Koffie RM, Hashimoto T, Tai HC, et al. (2012) Apolipoprotein E4 effects in Alzheimer's disease are mediated by synaptotoxic oligomeric amyloid-β. Brain : a Journal of Neurology. 135: 2155-68
de Calignon A, Polydoro M, Suárez-Calvet M, et al. (2012) Propagation of tau pathology in a model of early Alzheimer's disease. Neuron. 73: 685-97
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