Jill Lahti
Affiliations: | University of Tennessee Health Science Center, Memphis, TN, United States |
Area:
Molecular BiologyGoogle:
"Jill Lahti"
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Publications
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| Delehouzé C, Godl K, Loaëc N, et al. (2014) CDK/CK1 inhibitors roscovitine and CR8 downregulate amplified MYCN in neuroblastoma cells. Oncogene. 33: 5675-87 |
| Choi HH, Choi HK, Jung SY, et al. (2014) CHK2 kinase promotes pre-mRNA splicing via phosphorylating CDK11 p110 Oncogene. 33: 108-115 |
| Loyer P, Busson A, Trembley JH, et al. (2011) The RNA binding motif protein 15B (RBM15B/OTT3) is a functional competitor of serine-arginine (SR) proteins and antagonizes the positive effect of the CDK11p110-cyclin L2α complex on splicing. The Journal of Biological Chemistry. 286: 147-59 |
| Malumbres M, Harlow E, Hunt T, et al. (2009) Cyclin-dependent kinases: a family portrait. Nature Cell Biology. 11: 1275-6 |
| Sivakolundu SG, Nourse A, Moshiach S, et al. (2008) Intrinsically unstructured domains of Arf and Hdm2 form bimolecular oligomeric structures in vitro and in vivo. Journal of Molecular Biology. 384: 240-54 |
| Inoue A, Hyle J, Lechner MS, et al. (2008) Perturbation of HP1 localization and chromatin binding ability causes defects in sister-chromatid cohesion. Mutation Research. 657: 48-55 |
| Loyer P, Trembley JH, Grenet JA, et al. (2008) Characterization of cyclin L1 and L2 interactions with CDK11 and splicing factors: influence of cyclin L isoforms on splice site selection. The Journal of Biological Chemistry. 283: 7721-32 |
| Inoue A, Li T, Roby SK, et al. (2007) Loss of ChlR1 helicase in mouse causes lethality due to the accumulation of aneuploid cells generated by cohesion defects and placental malformation. Cell Cycle (Georgetown, Tex.). 6: 1646-54 |
| Hu D, Valentine M, Kidd VJ, et al. (2007) CDK11(p58) is required for the maintenance of sister chromatid cohesion. Journal of Cell Science. 120: 2424-34 |
| Parish JL, Rosa J, Wang X, et al. (2006) The DNA helicase ChlR1 is required for sister chromatid cohesion in mammalian cells. Journal of Cell Science. 119: 4857-65 |