David Marcus

Affiliations: 
Vanderbilt University, Nashville, TN 
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"David Marcus"
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Publications

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Marcus DJ, Bedse G, Gaulden AD, et al. (2020) Endocannabinoid Signaling Collapse Mediates Stress-Induced Amygdalo-Cortical Strengthening. Neuron
Henderson-Redmond AN, Nealon CM, Davis BJ, et al. (2019) c-Jun N terminal kinase signaling pathways mediate cannabinoid tolerance in an agonist-specific manner. Neuropharmacology. 107847
Berezniuk I, Rodriguiz RM, Zee ML, et al. (2017) ProSAAS-derived peptides are regulated by cocaine and are required for sensitization to the locomotor effects of cocaine. Journal of Neurochemistry
Marcus DJ, Henderson-Redmond AN, Gonek M, et al. (2017) Mice expressing a "hyper-sensitive" form of the CB1 cannabinoid receptor (CB1) show modestly enhanced alcohol preference and consumption. Plos One. 12: e0174826
Bluett RJ, Báldi R, Haymer A, et al. (2017) Endocannabinoid signalling modulates susceptibility to traumatic stress exposure. Nature Communications. 8: 14782
Marcus DJ, Zee ML, Davis BJ, et al. (2016) Mice Expressing a "Hyper-Sensitive" Form of the Cannabinoid Receptor 1 (CB1) Are Neither Obese Nor Diabetic. Plos One. 11: e0160462
Yuill MB, Zee ML, Marcus D, et al. (2016) Tolerance to the antinociceptive and hypothermic effects of morphine is mediated by multiple isoforms of c-Jun N-terminal kinase. Neuroreport
Marcus DJ, Zee M, Hughes A, et al. (2015) Tolerance to the antinociceptive effects of chronic morphine requires c-Jun N-terminal kinase. Molecular Pain. 11: 34
Morgan DJ, Davis BJ, Kearn CS, et al. (2014) Mutation of putative GRK phosphorylation sites in the cannabinoid receptor 1 (CB1R) confers resistance to cannabinoid tolerance and hypersensitivity to cannabinoids in mice. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 34: 5152-63
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