Elisenda Sanz
Affiliations: | Autonomous University of Barcelona, Spain |
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Publications
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Machuca-Márquez P, Sánchez-Benito L, Menardy F, et al. (2023) Vestibular CCK signaling drives motion sickness-like behavior in mice. Proceedings of the National Academy of Sciences of the United States of America. 120: e2304933120 |
Zagmutt S, Mera P, González-García I, et al. (2023) CPT1A in AgRP neurons is required for sex-dependent regulation of feeding and thirst. Biology of Sex Differences. 14: 14 |
Pauli JL, Chen JY, Basiri ML, et al. (2022) Molecular and anatomical characterization of parabrachial neurons and their axonal projections. Elife. 11 |
Gella A, Prada-Dacasa P, Carrascal M, et al. (2020) Mitochondrial Proteome of Affected Glutamatergic Neurons in a Mouse Model of Leigh Syndrome. Frontiers in Cell and Developmental Biology. 8: 660 |
Puighermanal E, Castell L, Esteve-Codina A, et al. (2020) Functional and molecular heterogeneity of D2R neurons along dorsal ventral axis in the striatum. Nature Communications. 11: 1957 |
Bolea I, Gella A, Sanz E, et al. (2019) Defined neuronal populations drive fatal phenotype in a mouse model of Leigh Syndrome. Elife. 8 |
Sanz E, Bean JC, Carey DP, et al. (2019) RiboTag: Ribosomal Tagging Strategy to Analyze Cell-Type-Specific mRNA Expression In Vivo. Current Protocols in Neuroscience. 88: e77 |
Bolea I, Gella A, Sanz E, et al. (2019) Author response: Defined neuronal populations drive fatal phenotype in a mouse model of Leigh syndrome Elife |
Chen B, Hui J, Montgomery KS, et al. (2017) Loss of Mitochondrial Ndufs4 in Striatal Medium Spiny Neurons Mediates Progressive Motor Impairment in a Mouse Model of Leigh Syndrome. Frontiers in Molecular Neuroscience. 10: 265 |
Rainwater A, Sanz E, Palmiter RD, et al. (2017) Striatal GPR88 modulates foraging efficiency. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience |