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Georg Köhr

Affiliations: 
Max Planck Institute, Stuttgart, Baden-Württemberg, Germany 
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"Georg Köhr"
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Parents

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Peter H. Seeburg post-doc

Children

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Patrick Jendritza research assistant MPI for Medical Research
Shi-Bin Li grad student MPI for Medical Research
Mario Treviño Villegas post-doc Max Planck Institute

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Publications

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Bertocchi I, Eltokhi A, Rozov A, et al. (2021) Author Correction: Voltage-independent GluN2A-type NMDA receptor Ca signaling promotes audiogenic seizures, attentional and cognitive deficits in mice. Communications Biology. 4: 187
Bertocchi I, Eltokhi A, Rozov A, et al. (2021) Voltage-independent GluN2A-type NMDA receptor Ca signaling promotes audiogenic seizures, attentional and cognitive deficits in mice. Communications Biology. 4: 59
Berberich S, Pohle J, Pollard M, et al. (2017) Interplay between global and pathway-specific synaptic plasticity in CA1 pyramidal cells. Scientific Reports. 7: 17040
Lissek T, Adams M, Adelman J, et al. (2017) Building Bridges through Science. Neuron. 96: 730-735
Bröker-Lai J, Kollewe A, Schindeldecker B, et al. (2017) Heteromeric channels formed by TRPC1, TRPC4 and TRPC5 define hippocampal synaptic transmission and working memory. The Embo Journal
Shimshek DR, Bus T, Schupp B, et al. (2017) Different Forms of AMPA Receptor Mediated LTP and Their Correlation to the Spatial Working Memory Formation. Frontiers in Molecular Neuroscience. 10: 214
Ballesteros JJ, Buschler A, Köhr G, et al. (2016) Afferent Input Selects NMDA Receptor Subtype to Determine the Persistency of Hippocampal LTP in Freely Behaving Mice. Frontiers in Synaptic Neuroscience. 8: 33
Elagabani MN, Briševac D, Kintscher M, et al. (2016) Subunit-Selective NMDA Receptor Signaling through BRAG1 and BRAG2 during Synapse Maturation. The Journal of Biological Chemistry
Li SB, Du D, Hasan MT, et al. (2014) D4 Receptor Activation Differentially Modulates Hippocampal Basal and Apical Dendritic Synapses in Freely Moving Mice. Cerebral Cortex (New York, N.Y. : 1991)
Punnakkal P, Jendritza P, Köhr G. (2012) Influence of the intracellular GluN2 C-terminal domain on NMDA receptor function. Neuropharmacology. 62: 1985-92
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