Hugues Abriel
Affiliations: | University of Bern, Bern, Bern, Switzerland |
Area:
ion channels, channelopathiesGoogle:
"Hugues Abriel"Mean distance: (not calculated yet)
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Publications
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Iamshanova O, Hämmerli AF, Ramaye E, et al. (2024) The dispensability of 14-3-3 proteins for the regulation of human cardiac sodium channel Nav1.5. Plos One. 19: e0298820 |
Arullampalam P, Essers MC, Boukenna M, et al. (2023) Knockdown of the TRPM4 channel alters cardiac electrophysiology and hemodynamics in a sex- and age-dependent manner in mice. Physiological Reports. 11: e15783 |
Selimi Z, Rougier JS, Abriel H, et al. (2023) A detailed analysis of single-channel Na 1.5 recordings does not reveal any cooperative gating. The Journal of Physiology. 601: 3847-3868 |
Boukenna M, Rougier JS, Aghagolzadeh P, et al. (2023) Multiomics uncover the proinflammatory role of deletion after myocardial infarction in mice. American Journal of Physiology. Heart and Circulatory Physiology. 324: H504-H518 |
Preti B, Rougier JS, Papapostolou I, et al. (2022) Targeting Ion Channel TRPM4. Chimia. 76: 1039-1044 |
Dokshokova L, Franzoso M, Di Bona A, et al. (2022) Nerve growth factor transfer from cardiomyocytes to innervating sympathetic neurons activates TrkA receptors at the neuro-cardiac junction. The Journal of Physiology |
Arullampalam P, Preti B, Ross-Kaschitza D, et al. (2021) Species-Specific Effects of Cation Channel TRPM4 Small-Molecule Inhibitors. Frontiers in Pharmacology. 12: 712354 |
Ozhathil LC, Rougier JS, Arullampalam P, et al. (2021) Deletion of Alters the Function of the Na1.5 Channel in Murine Cardiac Myocytes. International Journal of Molecular Sciences. 22 |
Hu Y, Kaschitza DR, Essers M, et al. (2021) Pathological activation of CaMKII induces arrhythmogenicity through TRPM4 overactivation. Pflugers Archiv : European Journal of Physiology |
Wang Z, Vermij SH, Sottas V, et al. (2020) Calmodulin binds to the N-terminal domain of the cardiac sodium channel Na1.5. Channels (Austin, Tex.). 14: 268-286 |